PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25573487-8	2015	On the other hand inhibition of elongation of N-glycosylation by tunicamycin (TM) resulted in inhibition of Phaseolus vulgaris-L (L-PHA) lectin-binding activity and inhibited cell adhesion to galectin-8, laminin and fibronectin.	Tunicamycin	65-76	fibronectin 1	Homo sapiens	216-227	
30017234-7	2018	ER stress inducer, tunicamycin (200 ng/mL) significantly increased the level of autophagy and reduced expression of fibronectin in HCFs, both of which were reversed by 4 Phenylbutyric acid.	Tunicamycin	19-30	fibronectin 1	Homo sapiens	116-127	
30017234-9	2018	LC3 co-localized with fibronectin when stimulated HCFs with tunicamycin.	Tunicamycin	60-71	fibronectin 1	Homo sapiens	22-33	
29344654-4	2018	Western blotting, immunohistochemistry, apoptotic assays and immunofluorescence were used to analyze the therapeutic effects of tunicamycin on apoptosis, growth, aggressiveness and cell cycle of colon tumor cells, by downregulation of fibronectin, vimentin and E-cadherin expression levels.	Tunicamycin	128-139	fibronectin 1	Homo sapiens	235-246	
31746423-10	2020	Treatment with TM or TG increased the expression of the ER stress markers glucose-regulated protein 78, phosphorylated eukaryotic initiation factor 2alpha, activating transcription factor (ATF)6, ATF4 and inositol-requiring protein 1alpha and the EMT markers fibronectin, vimentin, alpha-smooth muscle actin and neural cadherin.	Tunicamycin	15-17	fibronectin 1	Homo sapiens	259-270	
12845220-6	2003	Overproduction of fibronectin and tenascin by HMCs was normalized by tunicamycin and brefeldin A.	Tunicamycin	69-80	fibronectin 1	Homo sapiens	18-29	
12845220-7	2003	Similarly, when HMCs were exposed to exogenous TGF-beta, the increase in fibronectin was reversed by tunicamycin and brefeldin A.	Tunicamycin	101-112	fibronectin 1	Homo sapiens	73-84	
2946699-2	1986	Tunicamycin was used to produce carbohydrate-depleted fibronectin; it was synthesized by cultured fibroblasts.	Tunicamycin	0-11	fibronectin 1	Homo sapiens	54-65	
12232600-0	1996	The Effect of Tunicamycin on the Interaction of Fibronectin and HT1080 Cells.	Tunicamycin	14-25	fibronectin 1	Homo sapiens	48-59	
1576204-4	1992	Tunicamycin treatment, monitored by lectin-induced aggregation, drastically diminished cell adhesion to laminin and fibronectin, whereas cell binding to collagen IV was not affected.	Tunicamycin	0-11	fibronectin 1	Homo sapiens	116-127	
35165522-8	2022	Moreover in vitro experiments, ER stress induced by tunicamycin (TM) not only significantly increased the expression of GRP78 and CHOP, but also caused the epithelial to myofibroblast transformation (EMT) of renal tubular epithelial cells, evidenced by decreased expression of E-cadherin and increased expression of vimentin, and extracellular matrix (ECM) deposition, evidenced by increased expression of fibronectin (FN).	Tunicamycin	52-63	fibronectin 1	Homo sapiens	406-417	
3894594-8	1985	If the cultures are treated with tunicamycin, the astrocytes produce fibronectin that is unglycosylated, as shown by [3H]glucosamine labeling, and is neither sulfated nor phosphorylated as indicated by [35S]O4 and [32P]O4 labeling studies.	Tunicamycin	33-44	fibronectin 1	Homo sapiens	69-80	
6425062-10	1984	Exposure of the cells to tunicamycin, on the other hand, caused a prominent cytoplasmic accumulation of VIIIR:Ag and, within 96 h, led to the disappearance of most of the VIIIR:Ag-positive granules but did not affect the intracellular distribution of fibronectin.	Tunicamycin	25-36	fibronectin 1	Homo sapiens	251-262	
386885-4	1978	Loss of cell surface fibronectin as a result of viral transformation, or due to treatment of normal cells with tunicamycin, an inhibitor of protein glycosylation, may contribute to the reduced adhesion and altered morphology observed in these circumstances.	Tunicamycin	111-122	fibronectin 1	Homo sapiens	21-32