PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 2946699-2 1986 Tunicamycin was used to produce carbohydrate-depleted fibronectin; it was synthesized by cultured fibroblasts. Tunicamycin 0-11 fibronectin 1 Homo sapiens 54-65 12232600-0 1996 The Effect of Tunicamycin on the Interaction of Fibronectin and HT1080 Cells. Tunicamycin 14-25 fibronectin 1 Homo sapiens 48-59 1576204-4 1992 Tunicamycin treatment, monitored by lectin-induced aggregation, drastically diminished cell adhesion to laminin and fibronectin, whereas cell binding to collagen IV was not affected. Tunicamycin 0-11 fibronectin 1 Homo sapiens 116-127 35165522-8 2022 Moreover in vitro experiments, ER stress induced by tunicamycin (TM) not only significantly increased the expression of GRP78 and CHOP, but also caused the epithelial to myofibroblast transformation (EMT) of renal tubular epithelial cells, evidenced by decreased expression of E-cadherin and increased expression of vimentin, and extracellular matrix (ECM) deposition, evidenced by increased expression of fibronectin (FN). Tunicamycin 52-63 fibronectin 1 Homo sapiens 406-417 3894594-8 1985 If the cultures are treated with tunicamycin, the astrocytes produce fibronectin that is unglycosylated, as shown by [3H]glucosamine labeling, and is neither sulfated nor phosphorylated as indicated by [35S]O4 and [32P]O4 labeling studies. Tunicamycin 33-44 fibronectin 1 Homo sapiens 69-80 6425062-10 1984 Exposure of the cells to tunicamycin, on the other hand, caused a prominent cytoplasmic accumulation of VIIIR:Ag and, within 96 h, led to the disappearance of most of the VIIIR:Ag-positive granules but did not affect the intracellular distribution of fibronectin. Tunicamycin 25-36 fibronectin 1 Homo sapiens 251-262 30017234-7 2018 ER stress inducer, tunicamycin (200 ng/mL) significantly increased the level of autophagy and reduced expression of fibronectin in HCFs, both of which were reversed by 4 Phenylbutyric acid. Tunicamycin 19-30 fibronectin 1 Homo sapiens 116-127 31746423-10 2020 Treatment with TM or TG increased the expression of the ER stress markers glucose-regulated protein 78, phosphorylated eukaryotic initiation factor 2alpha, activating transcription factor (ATF)6, ATF4 and inositol-requiring protein 1alpha and the EMT markers fibronectin, vimentin, alpha-smooth muscle actin and neural cadherin. Tunicamycin 15-17 fibronectin 1 Homo sapiens 259-270 386885-4 1978 Loss of cell surface fibronectin as a result of viral transformation, or due to treatment of normal cells with tunicamycin, an inhibitor of protein glycosylation, may contribute to the reduced adhesion and altered morphology observed in these circumstances. Tunicamycin 111-122 fibronectin 1 Homo sapiens 21-32 30017234-9 2018 LC3 co-localized with fibronectin when stimulated HCFs with tunicamycin. Tunicamycin 60-71 fibronectin 1 Homo sapiens 22-33 29344654-4 2018 Western blotting, immunohistochemistry, apoptotic assays and immunofluorescence were used to analyze the therapeutic effects of tunicamycin on apoptosis, growth, aggressiveness and cell cycle of colon tumor cells, by downregulation of fibronectin, vimentin and E-cadherin expression levels. Tunicamycin 128-139 fibronectin 1 Homo sapiens 235-246 25573487-8 2015 On the other hand inhibition of elongation of N-glycosylation by tunicamycin (TM) resulted in inhibition of Phaseolus vulgaris-L (L-PHA) lectin-binding activity and inhibited cell adhesion to galectin-8, laminin and fibronectin. Tunicamycin 65-76 fibronectin 1 Homo sapiens 216-227 12845220-6 2003 Overproduction of fibronectin and tenascin by HMCs was normalized by tunicamycin and brefeldin A. Tunicamycin 69-80 fibronectin 1 Homo sapiens 18-29 12845220-7 2003 Similarly, when HMCs were exposed to exogenous TGF-beta, the increase in fibronectin was reversed by tunicamycin and brefeldin A. Tunicamycin 101-112 fibronectin 1 Homo sapiens 73-84