PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
6323131-4	1984	To determine whether this loss of LH/hCG receptors was due to accelerated receptor degradation, turnover measurements were made using tunicamycin to inhibit LH/hCG receptor synthesis.	Tunicamycin	134-145	hypertrichosis 2 (generalised, congenital)	Homo sapiens	160-163	
9139799-7	1997	In addition, tunicamycin-derived, nonglycosylated LHRs were present at the cell surface and exhibited a phenotype consistent with mature receptors due to their capability to mediate hCG-stimulated cAMP production as well as bind oLH with high affinity.	Tunicamycin	13-24	hypertrichosis 2 (generalised, congenital)	Homo sapiens	182-185