PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10725401-4	2000	Tunicamycin inhibition experiments showed that the apical polarity of VEGF(165) was independent of N-glycosylation.	Tunicamycin	0-11	vascular endothelial growth factor A	Homo sapiens	70-74	
21310902-6	2011	On stress induction, HTRA1 and VEGFA were upregulated in human fetal RPE cells treated by tunicamycin and dithiothreitol, but reduced after treatment by MG132.	Tunicamycin	90-101	vascular endothelial growth factor A	Homo sapiens	31-36	
14738568-8	2004	RESULTS: We found that glutamine deprivation and treatment with a chemical inducer of ER stress (tunicamycin) caused a marked induction of the secretion of both VEGF and IL-8 protein by a human breast adenocarcinoma cell line (TSE cells).	Tunicamycin	97-108	vascular endothelial growth factor A	Homo sapiens	161-165	
12147617-11	2002	Glucose deprivation or treatment of ARPE-19 cells with tunicamycin, brefeldin A, the calcium ionophore A23187, or thapsigargin increased the expression of VEGF mRNA in these cells by 8- to 10-fold.	Tunicamycin	55-66	vascular endothelial growth factor A	Homo sapiens	155-159	
1540178-5	1992	Preincubation of the transfected cells with the N-glycosylation inhibitor tunicamycin resulted in the conversion of the 47 kDa VEGF homodimer into a smaller, deglycosylated form of 42 kDa.	Tunicamycin	74-85	vascular endothelial growth factor A	Homo sapiens	127-131	
1930196-6	1991	When glycosylation was prevented by tunicamycin, individual bands of nonglycosylated vascular permeability factor were also secreted at equivalent rates, but much more slowly (approximately 60 min) than native glycoprotein.	Tunicamycin	36-47	vascular endothelial growth factor A	Homo sapiens	85-113	
23434931-4	2013	In lymphoblastoid cells induced to undergo endoplasmic reticulum (ER) stress by treatment of tunicamycin, higher fold change of TCF7L2 and VEGFA mRNA levels were observed in rs7903146-TT cells than in rs7903146-CC cells (P = 0.02 for TCF7L2; P = 0.004 for VEGFA), suggesting that ER stress plays a role in PDR pathogenesis.	Tunicamycin	93-104	vascular endothelial growth factor A	Homo sapiens	139-144	
23434931-4	2013	In lymphoblastoid cells induced to undergo endoplasmic reticulum (ER) stress by treatment of tunicamycin, higher fold change of TCF7L2 and VEGFA mRNA levels were observed in rs7903146-TT cells than in rs7903146-CC cells (P = 0.02 for TCF7L2; P = 0.004 for VEGFA), suggesting that ER stress plays a role in PDR pathogenesis.	Tunicamycin	93-104	vascular endothelial growth factor A	Homo sapiens	256-261	
27032713-4	2016	The ER stress inducer tunicamycin increased human chorionic gonadotropin-induced VEGFA production in human granulosa cells through the induction of XBP1(S), and pretreatment with the ER stress inhibitor tauroursodeoxycholic acid (TUDCA) abrogated the effect of tunicamycin.	Tunicamycin	22-33	vascular endothelial growth factor A	Homo sapiens	81-86