PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27313692-6	2016	Glucose uptake rate and lactate production assays demonstrated that miR-144 expression is decreased and therefore enhances the aerobic metabolism in lung cancer cells.	Lactic Acid	24-31	microRNA 144	Homo sapiens	68-75	
27313692-7	2016	In addition, western blot analysis revealed that miR-144 performs this function by increasing the expression of glucose transporter 1 (GLUT1), leading to an increase in glucose uptake and lactate production.	Lactic Acid	188-195	microRNA 144	Homo sapiens	49-56	
26662316-5	2016	As a result, cells overexpressing miR-144 exhibited a metabolic shift, including enhanced glucose uptake and lactate production.	Lactic Acid	109-116	microRNA 144	Homo sapiens	34-41	
32308422-13	2020	Functionally, FEZF1-AS1 silencing or miR-144 overexpression inhibited cell viability, the glucose and lactate productions and promoted cell apoptosis in Saos-2 and HOS cells.	Lactic Acid	102-109	microRNA 144	Homo sapiens	37-44	
32308422-14	2020	Furthermore, miR-144 inhibitor mitigated the inhibitory effects on cell viability, the glucose and lactate productions and the promoted effect on cell apoptosis rate in Saos-2 and HOS cells induced by FEZF1-AS1 depletion.	Lactic Acid	99-106	microRNA 144	Homo sapiens	13-20