PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23793235-7	2014	Transient transfection experiments showed that IL-6 induced the expression of a reporter gene under the control of a cAMP response element-like region in the human FAAH promoter.	Cyclic AMP	117-121	fatty acid amide hydrolase	Homo sapiens	164-168	
15606754-1	2004	We have shown recently that in human T lymphocytes, leptin stimulates activity and expression of the endocannabinoid-degrading enzyme fatty acid amide hydrolase (FAAH), through STAT3 (signal transducer and activator of transcription 3) and its CRE (cAMP response element)-like transcriptional target in the FAAH promoter [Maccarrone, M., Di Rienzo, M., Finazzi-Agro, A., & Rossi, A.	Cyclic AMP	249-253	fatty acid amide hydrolase	Homo sapiens	134-160	
15606754-1	2004	We have shown recently that in human T lymphocytes, leptin stimulates activity and expression of the endocannabinoid-degrading enzyme fatty acid amide hydrolase (FAAH), through STAT3 (signal transducer and activator of transcription 3) and its CRE (cAMP response element)-like transcriptional target in the FAAH promoter [Maccarrone, M., Di Rienzo, M., Finazzi-Agro, A., & Rossi, A.	Cyclic AMP	249-253	fatty acid amide hydrolase	Homo sapiens	162-166	
12556536-5	2003	Analysis of the FAAH promoter showed a cAMP-response element-like site, which is a transcriptional target of STAT3.	Cyclic AMP	39-43	fatty acid amide hydrolase	Homo sapiens	16-20	
12799380-8	2003	Furthermore, the effect of progesterone on FAAH promoter was additive to that of physiological amounts of leptin, which binds to a cAMP response element-like site in the promoter region.	Cyclic AMP	131-135	fatty acid amide hydrolase	Homo sapiens	43-47