PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31645561-4	2019	We find that interactions of the Galphas and Galphaq C termini with the beta2-adrenergic receptor (beta2-AR), targeted at the G-protein-binding site, enhance Gs activation and cyclic AMP levels.	Cyclic AMP	176-186	galactosamine (N-acetyl)-6-sulfatase	Homo sapiens	33-40	
33363467-9	2020	Additionally, GE could inhibit the release of inflammatory factors and suppress the expression of cAMP, which is the key factor of the conversion of Galphai and Galphas.	Cyclic AMP	98-102	galactosamine (N-acetyl)-6-sulfatase	Homo sapiens	161-168	
10946087-5	2000	However, GAS potentiated the PGE(1)-evoked cyclic AMP level in a bell-shaped, concentration-dependent manner.	Cyclic AMP	43-53	galactosamine (N-acetyl)-6-sulfatase	Homo sapiens	9-12	
10946087-10	2000	The results suggest that GAS played an additional role in potentiating the PGE(1)-induced cyclic AMP synthesis.	Cyclic AMP	90-100	galactosamine (N-acetyl)-6-sulfatase	Homo sapiens	25-28