PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30501137-2	2020	The results showed that the renal damage induced by CCl4 was associated with a rise in oxidative stress monitored by a significant increase of TBARS and PCO levels (+89% and +136% respectively, p < .001) and a significant decrease of GSH level (-68%, p < .001) and antioxidants enzymes such as SOD, CAT, and GPX activities (-41.7%, -47.8%, and -50.5%; p < .001, respectively).	Thiobarbituric Acid Reactive Substances	143-148	chemokine (C-C motif) ligand 4	Mus musculus	52-56	
29108440-2	2018	Administration of a single dose of CCl4 caused cardio toxicity as monitored by an increase in lipid peroxidation (thiobarbituric acid reactive substances), protein carbonyl level and antioxidant markers (superoxide dismutase, catalase, glutathione peroxidase, glutathione and vitamin C) in the heart tissue.	Thiobarbituric Acid Reactive Substances	114-153	chemokine (C-C motif) ligand 4	Mus musculus	35-39	
29636837-3	2018	The CCl4 treatment has significantly increased TBARS levels and reduced the antioxidant enzyme such as GSH, GPx, GR, GST, CAT, and SOD in group 2 compared to group 1, while the Zingerone treatment showed significant reduction in TBARS levels and increased the antioxidant enzymes in group 3 (CCl4 + Zingerone) as compared to group 2.	Thiobarbituric Acid Reactive Substances	47-52	chemokine (C-C motif) ligand 4	Mus musculus	4-8	
29636837-3	2018	The CCl4 treatment has significantly increased TBARS levels and reduced the antioxidant enzyme such as GSH, GPx, GR, GST, CAT, and SOD in group 2 compared to group 1, while the Zingerone treatment showed significant reduction in TBARS levels and increased the antioxidant enzymes in group 3 (CCl4 + Zingerone) as compared to group 2.	Thiobarbituric Acid Reactive Substances	229-234	chemokine (C-C motif) ligand 4	Mus musculus	4-8	
29080450-4	2018	The exposure to a single dose of CCl4 caused cardiotoxicity expressed by an increase in lipid peroxidation (TBARS), protein carbonyls (PC) levels and in antioxidant markers (superoxide dismutase (SOD), catalase (CAT), gluthathione peroxidase (GPx), glutathione (GSH) and Vitamin C levels) in the CCl4-treated group when compared with the untreated group.	Thiobarbituric Acid Reactive Substances	108-113	chemokine (C-C motif) ligand 4	Mus musculus	33-37	
30481781-12	2018	In an in vivo study, a thiobarbituric acid reactive substances assay showed that MRE inhibited CCl4-induced oxidative stress and expression of nitrotyrosine.	Thiobarbituric Acid Reactive Substances	23-62	chemokine (C-C motif) ligand 4	Mus musculus	95-99	
20589743-4	2010	Our results revealed that MIL glycoprotein reduced the activities of ALT, LDH and TBARS in serum from CCl(4) -treated mice.	Thiobarbituric Acid Reactive Substances	82-87	chemokine (C-C motif) ligand 4	Mus musculus	102-108	
16823256-4	2006	When mice were treated with CCl4 in the absence of UDN glycoprotein, the activities of ALT, LDH, and TBARS were increased, while the antioxidant enzymes activities were decreased.	Thiobarbituric Acid Reactive Substances	101-106	chemokine (C-C motif) ligand 4	Mus musculus	28-32	
16823256-5	2006	However, when the mice were treated with CCl4 in the presence of UDN glycoprotein, the activities of ALT, LDH, and TBARS were significantly reduced and SOD, CAT, and GPx activities were remarkably increased.	Thiobarbituric Acid Reactive Substances	115-120	chemokine (C-C motif) ligand 4	Mus musculus	41-45	
11824561-4	2002	These extracts also inhibited CCl4-induced thiobarbituric acid-reactive substance (TBA-RS) formation, which indicates increased lipid peroxidation in the liver.	Thiobarbituric Acid Reactive Substances	43-81	chemokine (C-C motif) ligand 4	Mus musculus	30-34	
11824561-4	2002	These extracts also inhibited CCl4-induced thiobarbituric acid-reactive substance (TBA-RS) formation, which indicates increased lipid peroxidation in the liver.	Thiobarbituric Acid Reactive Substances	83-89	chemokine (C-C motif) ligand 4	Mus musculus	30-34	
9438234-3	1997	When thiobarbituric acid-reactive substances (TBARS) were measured in the absence of antioxidant the FO diet and CCl4 treatment markedly increased liver TBARS values synergistically, apparently supporting the interpretation that the highly autoxidizable DHA accelerates lipid peroxidation induced by CCl4.	Thiobarbituric Acid Reactive Substances	5-44	chemokine (C-C motif) ligand 4	Mus musculus	113-117	
9438234-3	1997	When thiobarbituric acid-reactive substances (TBARS) were measured in the absence of antioxidant the FO diet and CCl4 treatment markedly increased liver TBARS values synergistically, apparently supporting the interpretation that the highly autoxidizable DHA accelerates lipid peroxidation induced by CCl4.	Thiobarbituric Acid Reactive Substances	153-158	chemokine (C-C motif) ligand 4	Mus musculus	113-117	
1606637-2	1992	administration of an EtOH extract of the stems of Kadsura heteroclita (Schizandraceae) or its major constituent, kadsurin, resulted in significant decreases of CCl4-induced lipid-peroxidation products, such as thiobarbituric acid reactive substances (TBA-RS), conjugated dienes and fluorescent products in the liver of mice.	Thiobarbituric Acid Reactive Substances	210-249	chemokine (C-C motif) ligand 4	Mus musculus	160-164	
1606637-2	1992	administration of an EtOH extract of the stems of Kadsura heteroclita (Schizandraceae) or its major constituent, kadsurin, resulted in significant decreases of CCl4-induced lipid-peroxidation products, such as thiobarbituric acid reactive substances (TBA-RS), conjugated dienes and fluorescent products in the liver of mice.	Thiobarbituric Acid Reactive Substances	251-257	chemokine (C-C motif) ligand 4	Mus musculus	160-164	
1798788-1	1991	Intraperitoneal administration of CCl4 to mice led to significant increases of thiobarbituric acid-reactive substances (TBA-RS), free malondialdehyde (MDA), lipid conjugated dienes and fluorescent lipid peroxidation products in the liver.	Thiobarbituric Acid Reactive Substances	120-126	chemokine (C-C motif) ligand 4	Mus musculus	34-38