PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30468467-0	2018	Overexpression of miR-215-3p sensitizes colorectal cancer to 5-fluorouracil induced apoptosis through regulating CXCR1.	Fluorouracil	61-75	microRNA 215	Homo sapiens	18-25	
32922964-10	2020	Polymorphisms of the glucocorticoid receptor and variants of CCL2, YAP1, miR-21-5p and NF-kappabeta might be responsible for different responses to treatments used in keloid scars such as 5-fluorouracil.	Fluorouracil	188-202	microRNA 215	Homo sapiens	73-82	
31542354-0	2019	Inhibition of hydrogen sulfide synthesis reverses acquired resistance to 5-FU through miR-215-5p-EREG/TYMS axis in colon cancer cells.	Fluorouracil	73-77	microRNA 215	Homo sapiens	86-93	
30590435-13	2019	Melatonin may serve as a potential therapeutic option on its own, or in conjunction with 5-FU, in the treatment of patients with advanced or chemoresistant CRC.Melatonin inhibits the growth of 5-FU resistant colorectal cancer (CRC) cells through upregulation of miR-215-5p and a concomitant downregulation of TYMS.	Fluorouracil	193-197	microRNA 215	Homo sapiens	262-269	
30468467-6	2018	RESULTS: miR-215-3p was down-expressed in the 5-FU resistant cell compared to the parent cell.	Fluorouracil	46-50	microRNA 215	Homo sapiens	9-16	
30468467-8	2018	Furthermore, miR-215-3p accelerated the apoptosis of colorectal cancer cell which was treated with 5-Fu.	Fluorouracil	99-103	microRNA 215	Homo sapiens	13-20	
30468467-9	2018	Mechanically, miR-215-3p regulated the level of endogenous CXCR1 in HCT116 cell and alternation of CXCR1 affected the 5-Fu sensibility mediated by miR-215-3p.	Fluorouracil	118-122	microRNA 215	Homo sapiens	14-21	
30468467-9	2018	Mechanically, miR-215-3p regulated the level of endogenous CXCR1 in HCT116 cell and alternation of CXCR1 affected the 5-Fu sensibility mediated by miR-215-3p.	Fluorouracil	118-122	microRNA 215	Homo sapiens	147-154	
20647341-0	2010	miR-192/miR-215 influence 5-fluorouracil resistance through cell cycle-mediated mechanisms complementary to its post-transcriptional thymidilate synthase regulation.	Fluorouracil	26-40	microRNA 215	Homo sapiens	8-15	
23532818-7	2013	It seemed that patients with high expressions of miR-215 could benefit from 5-fluorouracil-containing adjuvant chemotherapy without significant difference, whereas this phenomenon was reverse in patients with low expressions of miR-215.	Fluorouracil	76-90	microRNA 215	Homo sapiens	49-56