PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34589581-10	2021	Finally, in vitro and in vivo xenograft experiments consistently demonstrated that inhibition of EGFR by the specific inhibitor erlotinib effectively enhanced the anti-tumor toxicity of 5-Fu by targeting the EGFR-FGD5-AS1-miR-330-3p-HK2 pathway.	Fluorouracil	186-190	microRNA 330	Homo sapiens	222-229	
34589581-0	2021	Targeting EGFR sensitizes 5-Fu-resistant colon cancer cells through modification of the lncRNA-FGD5-AS1-miR-330-3p-Hexokinase 2 axis.	Fluorouracil	26-30	microRNA 330	Homo sapiens	104-111	
28521444-6	2017	Ectopic miR-330 expression decreased cell proliferation and enhanced cell chemosensitivity to 5-FU via the cell apoptosis pathway in CRC.	Fluorouracil	94-98	microRNA 330	Homo sapiens	8-15	
34589581-9	2021	Rescue experiments demonstrated that FGD5-AS1 promotes glycolysis through modulating the miR-330-3p-HK2 axis, leading to 5-Fu resistance of CRC cancer cells.	Fluorouracil	121-125	microRNA 330	Homo sapiens	89-96	
35609416-4	2022	Notably, ectopic expression of miR-330-5p restrained tumor cell proliferation, migration, and enhance the sensitivity of CRC cells to 5-FU.	Fluorouracil	134-138	microRNA 330	Homo sapiens	31-38	
28521444-9	2017	In conclusion, the results of the present study indicated that miR-330 targeted TYMS to inhibit the proliferation and enhance the chemosensitivity of CRC cells to 5-FU by promoting cell apoptosis.	Fluorouracil	163-167	microRNA 330	Homo sapiens	63-70