PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31077347-0	2019	Spermidine-enhanced autophagic flux improves cardiac dysfunction following myocardial infarction by targeting the AMPK/mTOR signalling pathway.	Spermidine	0-10	mechanistic target of rapamycin kinase	Rattus norvegicus	119-123	
31077347-11	2019	The protective effects of spermidine on cardiomyocyte apoptosis and cardiac dysfunction were abolished by the autophagy inhibitor chloroquine, indicating that spermidine exerted cardioprotective effects at least partly through promoting autophagic flux, by activating the AMPK/mTOR signalling pathway.	Spermidine	26-36	mechanistic target of rapamycin kinase	Rattus norvegicus	277-281	
31077347-11	2019	The protective effects of spermidine on cardiomyocyte apoptosis and cardiac dysfunction were abolished by the autophagy inhibitor chloroquine, indicating that spermidine exerted cardioprotective effects at least partly through promoting autophagic flux, by activating the AMPK/mTOR signalling pathway.	Spermidine	159-169	mechanistic target of rapamycin kinase	Rattus norvegicus	277-281	
31077347-12	2019	CONCLUSIONS AND IMPLICATIONS: Our findings suggest that spermidine improved MI-induced cardiac dysfunction by promoting AMPK/mTOR-mediated autophagic flux.	Spermidine	56-66	mechanistic target of rapamycin kinase	Rattus norvegicus	125-129