PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12198703-0	2002	JunD stabilization results in inhibition of normal intestinal epithelial cell growth through P21 after polyamine depletion.	Polyamines	103-112	H3 histone pseudogene 16	Homo sapiens	93-96	
20095972-5	2009	Specific PAHAs (polyaminohydroxamic acids) and PABAs (polyaminobenzamides) polyamine analogues have demonstrated potent inhibition of the HDACs, re-expression of p21 and significant inhibition of tumour growth.	Polyamines	75-84	H3 histone pseudogene 16	Homo sapiens	162-165	
18548481-11	2008	CONCLUSIONS: The pADxsi-PSES-AdoMetDC-ODC-PolyA AV specifically inhibited the expression of ODC and AdoMetDC and the synthesis of polyamine, while it induced p21 expression, resulting in cell growth arrest in the G1 phase in prostate cancer cells but not in other cells.	Polyamines	130-139	H3 histone pseudogene 16	Homo sapiens	158-161	
12198703-9	2002	Growth arrest following polyamine depletion also was accompanied by increases in both p21 expression and its promoter activity.	Polyamines	24-33	H3 histone pseudogene 16	Homo sapiens	86-89	
12198703-12	2002	CONCLUSIONS: Polyamines negatively regulate junD gene expression posttranscriptionally, and increased JunD/AP-1 inhibits intestinal epithelial cell proliferation at least partially through the activation of p21 promoter.	Polyamines	13-23	H3 histone pseudogene 16	Homo sapiens	207-210	
10047792-0	1998	Polyamine analogue-mediated cell cycle responses in human melanoma cells involves the p53, p21, Rb regulatory pathway.	Polyamines	0-9	H3 histone pseudogene 16	Homo sapiens	91-94