PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18834226-3	2008	OBJECTIVE: To investigate and compare the molecular and clinicopathologic features of CIMP-positive colorectal cancers defined by classic (p16, hMLH1, MINT1, MINT2, MINT31) and new (CACNA1G, IGF2, NEUROG1, RUNX3, SOCS1) CIMP panels.	Cyclic IMP	86-90	RUNX family transcription factor 3	Homo sapiens	206-211	
18628431-6	2008	Tumors were classified as CIMP positive when at least three of five markers (RUNX3, CACNA1G, SOCS1, NEUROG1, and IGF2) were methylated according to MethyLight analysis.	Cyclic IMP	26-30	RUNX family transcription factor 3	Homo sapiens	77-82	
28542846-3	2017	CIMP status was determined by analyzing the 5-markers CAGNA1G, IGF2, NEUROG1, RUNX3 and SOCS1 by quantitative methylation specific PCR (qMSP).	Cyclic IMP	0-4	RUNX family transcription factor 3	Homo sapiens	78-83	
17898258-8	2007	The rates for the CIMP-related genes were 25% for RUNX3, 5% for NEUROG1 and CACNA1G, and 0 for SOCS-1 and IGF-2.	Cyclic IMP	18-22	RUNX family transcription factor 3	Homo sapiens	50-55	
17270239-6	2007	After stratification by sex, location, tumor differentiation, RUNX3 status, KRAS/BRAF status, or p53 status, CIMP-high was persistently correlated with TGFBR2 mutation.	Cyclic IMP	109-113	RUNX family transcription factor 3	Homo sapiens	62-67	
21791485-7	2012	The promoter regions of subset of genes highly specific to characterize the CIMP status (NEUROG1, IGF2, RUNX3, SOCS1, including MLH1) were hypermethylated, suggesting the presence of CIMP+ and MSI high tumor.	Cyclic IMP	76-80	RUNX family transcription factor 3	Homo sapiens	104-109	
26702772-5	2016	CIMP status was determined using the DNA methylation status of CACNA1G, IGF2, NEUROG1, RUNX3, and SOCS1.	Cyclic IMP	0-4	RUNX family transcription factor 3	Homo sapiens	87-92	
21068132-8	2011	The CIMP marker RUNX3 was the earliest CpG island showing significant change, followed by the CIMP markers NEUROG1 and CACNA1G at the hyperplastic polyp stage.	Cyclic IMP	4-8	RUNX family transcription factor 3	Homo sapiens	16-21