PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30585644-8	2019	RESULTS: Both HEDP and IP6 treatment triggered TGF-beta release and cell migration.	Phytic Acid	23-26	transforming growth factor beta 1	Homo sapiens	47-55	
30585644-12	2019	CONCLUSIONS: IP6 and HEDP were effective chelating agents on TGF-beta release, and cell migration was as effective as EDTA.	Phytic Acid	13-16	transforming growth factor beta 1	Homo sapiens	61-69	
23614294-0	2013	Induction of the expression of genes encoding TGF-beta isoforms and their receptors by inositol hexaphosphate in human colon cancer cells.	Phytic Acid	87-109	transforming growth factor beta 1	Homo sapiens	46-54	
23614294-7	2013	The aim of this study was to examine the effect of IP6 on the expression of genes encoding TGF-beta1, TGF-beta2, TGF-beta3 isoforms and their receptors TbetaRI, TbetaRII, TbetaRIII in human colorectal cancer cell line Caco-2.	Phytic Acid	51-54	transforming growth factor beta 1	Homo sapiens	91-100	
23614294-11	2013	At concentrations up to 1 mM, IP6 over-expressed the genes in 6 h lasting cultures, and its higher dose (2.5 mM) caused successively increasing transcript level of TGF-beta isoforms and receptors with the duration of experiment up to 12 h. The findings of this study indicate that one of anti-cancer abilities of IP6 can be realized by enhancing the gene expression of TGF-beta isoforms and their receptors at the transcriptional level.	Phytic Acid	30-33	transforming growth factor beta 1	Homo sapiens	164-172	
23614294-11	2013	At concentrations up to 1 mM, IP6 over-expressed the genes in 6 h lasting cultures, and its higher dose (2.5 mM) caused successively increasing transcript level of TGF-beta isoforms and receptors with the duration of experiment up to 12 h. The findings of this study indicate that one of anti-cancer abilities of IP6 can be realized by enhancing the gene expression of TGF-beta isoforms and their receptors at the transcriptional level.	Phytic Acid	30-33	transforming growth factor beta 1	Homo sapiens	369-377	
23614294-11	2013	At concentrations up to 1 mM, IP6 over-expressed the genes in 6 h lasting cultures, and its higher dose (2.5 mM) caused successively increasing transcript level of TGF-beta isoforms and receptors with the duration of experiment up to 12 h. The findings of this study indicate that one of anti-cancer abilities of IP6 can be realized by enhancing the gene expression of TGF-beta isoforms and their receptors at the transcriptional level.	Phytic Acid	313-316	transforming growth factor beta 1	Homo sapiens	164-172