PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 12663518-11 2003 At higher doses and longer treatment times, IP6 caused a marked increase in apoptosis, which was accompanied by increased levels of cleaved PARP and active caspase 3. Phytic Acid 44-47 poly(ADP-ribose) polymerase 1 Homo sapiens 140-144 19920184-3 2009 IP6 treatment of cells suppressed proliferation, induced apoptosis along with caspase-3 and poly(ADP-ribose) polymerase (PARP) cleavage, and inhibited constitutive activation of Akt and its upstream regulators PI3K, phosphoinositide-dependent kinase-1 and integrin-linked kinase-1 (ILK1). Phytic Acid 0-3 poly(ADP-ribose) polymerase 1 Homo sapiens 92-119 19920184-3 2009 IP6 treatment of cells suppressed proliferation, induced apoptosis along with caspase-3 and poly(ADP-ribose) polymerase (PARP) cleavage, and inhibited constitutive activation of Akt and its upstream regulators PI3K, phosphoinositide-dependent kinase-1 and integrin-linked kinase-1 (ILK1). Phytic Acid 0-3 poly(ADP-ribose) polymerase 1 Homo sapiens 121-125 11217149-11 2001 Phytic acid, an iron chelator, catalase, and superoxide dismutase each decreased WI-26 cell PARP activation caused by asbestos and CSE. Phytic Acid 0-11 poly(ADP-ribose) polymerase 1 Homo sapiens 92-96