PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12663518-11	2003	At higher doses and longer treatment times, IP6 caused a marked increase in apoptosis, which was accompanied by increased levels of cleaved PARP and active caspase 3.	Phytic Acid	44-47	poly(ADP-ribose) polymerase 1	Homo sapiens	140-144	
19920184-3	2009	IP6 treatment of cells suppressed proliferation, induced apoptosis along with caspase-3 and poly(ADP-ribose) polymerase (PARP) cleavage, and inhibited constitutive activation of Akt and its upstream regulators PI3K, phosphoinositide-dependent kinase-1 and integrin-linked kinase-1 (ILK1).	Phytic Acid	0-3	poly(ADP-ribose) polymerase 1	Homo sapiens	92-119	
19920184-3	2009	IP6 treatment of cells suppressed proliferation, induced apoptosis along with caspase-3 and poly(ADP-ribose) polymerase (PARP) cleavage, and inhibited constitutive activation of Akt and its upstream regulators PI3K, phosphoinositide-dependent kinase-1 and integrin-linked kinase-1 (ILK1).	Phytic Acid	0-3	poly(ADP-ribose) polymerase 1	Homo sapiens	121-125	
11217149-11	2001	Phytic acid, an iron chelator, catalase, and superoxide dismutase each decreased WI-26 cell PARP activation caused by asbestos and CSE.	Phytic Acid	0-11	poly(ADP-ribose) polymerase 1	Homo sapiens	92-96