PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10671943-14	2000	CONCLUSION: We conclude that in children, as in adults, body fat is the primary determinant for the circulating level of GHBP, and that the difference in body fat is probably the main factor for the higher levels of serum GHBP in girls compared with boys, as well as for the negative influence of testosterone levels in boys and of oestrogen levels in girls.	Testosterone	297-309	growth hormone receptor	Homo sapiens	222-226	
1767744-3	1991	In males, acceleration of growth results primarily from enhanced sensitivity of the GH-receptor-IGF I system to GH brought about by testosterone.	Testosterone	132-144	growth hormone receptor	Homo sapiens	84-95	
7714101-9	1995	Thus, serum GHBP is normal in hypogonadal men but is reduced by testosterone treatment irrespective of endogenous GH-secretory status.	Testosterone	64-76	growth hormone receptor	Homo sapiens	12-16	
7714101-10	1995	It was concluded that the effect of testosterone on GHBP is pharmacological and occurs independent of GH mediation.	Testosterone	36-48	growth hormone receptor	Homo sapiens	52-56	
1557221-12	1992	GH and testosterone have an opposite role in the regulation of the high affinity GH-BP.	Testosterone	7-19	growth hormone receptor	Homo sapiens	81-86	
1767744-6	1991	Though the pygmy data certainly supports a relationship between testosterone and the GH-receptor-IGF I axis, the undisputed tall stature of eunuchs remains a puzzle.	Testosterone	64-76	growth hormone receptor	Homo sapiens	85-96	
26031356-4	2015	EXPERIMENTAL APPROACH: The regulation of CYP2D and brain-enriched miRNAs by testosterone was investigated using SH-SY5Y cells, U251 cells, and HepG2 cells as well as orchiectomized growth hormone receptor knockout (GHR-KO) mice and rats.	Testosterone	76-88	growth hormone receptor	Homo sapiens	181-204