PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2156871-4	1990	In fact, the GM-CSF-dependent proliferation of AML-193 cells is strongly reduced in the presence of the amiloride analog EIPA, a specific inhibitor of the Na+/H+ exchanger.	Amiloride	104-113	colony stimulating factor 2	Homo sapiens	13-19	
8473054-8	1993	Pre-treatment of cells for 2 hr with non-toxic concentrations of pertussis toxin (PT) or amiloride resulted in a 50% inhibition of chemotaxis to GM-CSF.	Amiloride	89-98	colony stimulating factor 2	Homo sapiens	145-151	
8473054-9	1993	Therefore, GM-CSF, through PT- and amiloride-sensitive signal pathways, is a potent attractant for melanoma cells, the response to which is additive to that of other attractants.	Amiloride	35-44	colony stimulating factor 2	Homo sapiens	11-17	
1315776-10	1992	Third, the two amiloride derivatives 5-(N,N-hexamethylene) and 5-(N-ethyl-N-isopropyl)amiloride that specifically block the function of the Na+/H+ antiport also revert the protective effect of GM-CSF, IL-3, and TPA on MO7-E cells.	Amiloride	15-24	colony stimulating factor 2	Homo sapiens	193-199	
1646047-9	1991	The sodium channel blocker amiloride prevented the GM-CSF-induced change in pH, but did not affect the stimulation of c-myc transcription by GM-CSF.	Amiloride	27-36	colony stimulating factor 2	Homo sapiens	51-57	
7512958-5	1994	We have probed the pathways activated by SF and GM-CSF in suppression of active cell death (apoptosis) using two classes of inhibitors: Tyrphostins that are specific inhibitors of protein tyrosine kinase, and amiloride derivatives (5-(N,N-ethyl-n-isopropyl)amiloride and 5-(N,N-hexamethylene)amiloride) that have been designed as specific inhibitors of the Na+/H+ antiporter.	Amiloride	209-218	colony stimulating factor 2	Homo sapiens	48-54	
2154185-6	1990	Both agonists do, however, share an ability to activate an amiloride-sensitive Na+/H+ antiport and, furthermore, amiloride analogues are shown to inhibit the proliferative effects of GM-CSF on these cells.	Amiloride	59-68	colony stimulating factor 2	Homo sapiens	183-189	
2154185-6	1990	Both agonists do, however, share an ability to activate an amiloride-sensitive Na+/H+ antiport and, furthermore, amiloride analogues are shown to inhibit the proliferative effects of GM-CSF on these cells.	Amiloride	113-122	colony stimulating factor 2	Homo sapiens	183-189	
2471189-3	1989	Changes produced by GM-CSF are totally inhibited by amiloride and are significantly reduced in pertussis toxin-treated cells.	Amiloride	52-61	colony stimulating factor 2	Homo sapiens	20-26