PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
3066348-3	1988	We found that AlCl3 has a partial insulin-like effect on glucose transport activity (3-O-methylglucose uptake, expressed as % of equilibrium value per 4 s: basal 9.6 +/- 2, AlCl3 29.6 +/- 4, insulin 74.0 +/- 3).	Aluminum Chloride	14-19	insulin	Homo sapiens	34-41	
3066348-3	1988	We found that AlCl3 has a partial insulin-like effect on glucose transport activity (3-O-methylglucose uptake, expressed as % of equilibrium value per 4 s: basal 9.6 +/- 2, AlCl3 29.6 +/- 4, insulin 74.0 +/- 3).	Aluminum Chloride	14-19	insulin	Homo sapiens	191-198	
3066348-7	1988	Both substances acted faster than insulin itself (maximal values within 1 min for PLC, 2 min for AlCl3 and 5-10 min for insulin).	Aluminum Chloride	97-102	insulin	Homo sapiens	34-41	
3066348-10	1988	IN CONCLUSION: (1) the data show that AlCl3, probably through activation of a pertussis-toxin-inhibitable G protein, and PLC are able to modulate the intrinsic glucose carrier activity; (2) as pertussis toxin did not modify the effect of insulin, it seems unlikely that the insulin signal on glucose transport involves activation of this specific G protein.	Aluminum Chloride	38-43	insulin	Homo sapiens	238-245	
3066348-10	1988	IN CONCLUSION: (1) the data show that AlCl3, probably through activation of a pertussis-toxin-inhibitable G protein, and PLC are able to modulate the intrinsic glucose carrier activity; (2) as pertussis toxin did not modify the effect of insulin, it seems unlikely that the insulin signal on glucose transport involves activation of this specific G protein.	Aluminum Chloride	38-43	insulin	Homo sapiens	274-281	
32383521-0	2020	Biotin, coenzyme Q10, and their combination ameliorate aluminium chloride-induced Alzheimer"s disease via attenuating neuroinflammation and improving brain insulin signaling.	Aluminum Chloride	55-73	insulin	Homo sapiens	156-163	
32383521-5	2020	Coenzyme Q10 (CoQ10) and biotin possess antioxidant and anti-inflammatory properties, and, in this study, their impact on insulin signaling is investigated in an aluminium chloride (AlCl3 ) model of AD.	Aluminum Chloride	162-180	insulin	Homo sapiens	122-129	
32383521-10	2020	AlCl3 resulted in impaired memory, significant increase in Abeta, lipid peroxides, inflammatory markers, caspase-3, and pSer-IRS-1, with significant reduction of the antioxidants, pTyr-IRS-1, and p-Akt reflecting Abeta-induced inflammation and defective insulin signaling.	Aluminum Chloride	0-5	insulin	Homo sapiens	254-261