PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23266327-3	2013	Considering that activation of the extracellular signal-regulated protein kinase (ERK) is suggested to be involved in cocaine-induced behavioral sensitization, we study the effects of perinatal protein deprivation on phosphorylated ERK2 (pERK2) protein levels in the NAc (core and shell) during different drug-free withdrawal periods.	nac	267-270	Eph receptor B1	Rattus norvegicus	82-85	
27382351-8	2016	TNF-alpha induced ROS formation, phosphorylation of NF-kappaB p65 and ERK, and LOX-1 expression, which were suppressed by rotenone, DPI, NAC, and CPT.	nac	137-140	Eph receptor B1	Rattus norvegicus	70-73	
27382351-10	2016	CPT and NAC suppressed TNF-alpha-induced LOX-1 expression and phosphorylation of NF-kappaB p65 and ERK in rat aorta.	nac	8-11	Eph receptor B1	Rattus norvegicus	99-102	
23288619-8	2013	However, this effect was blocked by NAC (5 mM; ROS inhibitor), indicating that AGEs regulate MEK/ERK phosphorylation via ROS.	nac	36-39	Eph receptor B1	Rattus norvegicus	97-100	
18588920-7	2008	The effect was blocked by the antagonist (Nphe(1))nociceptin(1-13)NH(2) and attenuated by U0126, suggesting that the activation of ERK pathways is involved in the OFQ-induced nociceptive effect in the NAc of rats.	nac	201-204	Eph receptor B1	Rattus norvegicus	131-134	
34363828-2	2021	We administered morphine (10 mg/kg; MOR) to induce locomotor sensitization and ERK activation in the VTA and NAc.	nac	109-112	Eph receptor B1	Rattus norvegicus	79-82