PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7720780-5	1995	The cellular accumulation of [14C]pristinamycin IA was very low and was increased by P-glycoprotein inhibitors (verapamil, chlorpromazine and reserpine).	Carbon-14	30-33	ATP binding cassette subfamily B member 1	Homo sapiens	85-99	
12909563-0	2003	Measurement of hepatic and intestinal CYP3A4 and PGP activity by combined po + iv [14C]erythromycin breath and urine test.	Carbon-14	83-86	ATP binding cassette subfamily B member 1	Homo sapiens	49-52	
32707710-6	2020	Furthermore, XH-14C could stimulate the ATPase activity of ABCB1 and the molecular dynamic simulation revealed a stable binding pose of XH-14C-ABCB1 complex.	Carbon-14	16-19	ATP binding cassette subfamily B member 1	Homo sapiens	59-64	
32707710-6	2020	Furthermore, XH-14C could stimulate the ATPase activity of ABCB1 and the molecular dynamic simulation revealed a stable binding pose of XH-14C-ABCB1 complex.	Carbon-14	16-19	ATP binding cassette subfamily B member 1	Homo sapiens	143-148	
10665657-7	1999	The inhibition of [14C]docetaxel transport by the MDR modulators, as well as daunorubicin and vinblastine, was also found in LLC-PK1 cells, which endogenously express P-gp at lower levels, and concentrations showing similar levels of inhibition were lower than those in the case of LLC-GA5-COL150 cells.	Carbon-14	19-22	ATP binding cassette subfamily B member 1	Homo sapiens	167-171	
10381759-5	1999	The uptake of [14C]HSR-903 by multidrug-resistant K562/ADM cells, which express P-glycoprotein (P-gp), was significantly lower than that by the drug-sensitive parent K562 cells.	Carbon-14	15-18	ATP binding cassette subfamily B member 1	Homo sapiens	80-94	
10381759-5	1999	The uptake of [14C]HSR-903 by multidrug-resistant K562/ADM cells, which express P-glycoprotein (P-gp), was significantly lower than that by the drug-sensitive parent K562 cells.	Carbon-14	15-18	ATP binding cassette subfamily B member 1	Homo sapiens	96-100	
10100591-9	1999	Increased efflux of [14C]-Ro 32-2241 was seen with the more resistant KB-8-5-11 cells (although the percentage effluxed was very low as compared with [3H]-daunomycin), suggesting that Ro 32-2241 can act as a substrate for Pgp.	Carbon-14	21-24	ATP binding cassette subfamily B member 1	Homo sapiens	222-225	
9066677-8	1997	Incubation of MDR cells with [14C] N-ethylmaleimide showed that P-gp is directly modified at several Cysteine residues, as found from a complete proteolytic digestion of [14C] Nethylmaleimide labeled P-gp.	Carbon-14	30-33	ATP binding cassette subfamily B member 1	Homo sapiens	64-68	
9066677-8	1997	Incubation of MDR cells with [14C] N-ethylmaleimide showed that P-gp is directly modified at several Cysteine residues, as found from a complete proteolytic digestion of [14C] Nethylmaleimide labeled P-gp.	Carbon-14	30-33	ATP binding cassette subfamily B member 1	Homo sapiens	200-204	
9066677-8	1997	Incubation of MDR cells with [14C] N-ethylmaleimide showed that P-gp is directly modified at several Cysteine residues, as found from a complete proteolytic digestion of [14C] Nethylmaleimide labeled P-gp.	Carbon-14	171-174	ATP binding cassette subfamily B member 1	Homo sapiens	64-68	
9794492-3	1998	RESULTS: The basolateral to apical (B-A) flux of 10 microM [14C]-K02 across MDR1-MDCK cells was markedly greater than its apical to basolateral (A-B) flux (ratio = 39).	Carbon-14	60-63	ATP binding cassette subfamily B member 1	Homo sapiens	76-80