PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 7858064-5 1994 Treatment of FS-4 cells with sodium arsenite led to a very strong increase in the phosphorylation of Hsp28 demonstrable after 5 min and persisting for at least 4 h. Tyrosine phosphorylation of pp42 and pp44 MAP kinases was increased by TNF treatment, whereas arsenite produced a modest increase in tyrosine phosphorylation of pp44 while decreasing that of pp42 MAP kinase. sodium arsenite 29-44 tumor necrosis factor Homo sapiens 236-239 1417842-0 1992 The adenovirus E3 region 14.7 kDa protein, heat and sodium arsenite inhibit the TNF-induced release of arachidonic acid. sodium arsenite 52-67 tumor necrosis factor Homo sapiens 80-83 1417842-1 1992 In this report we show that the adenovirus E3 region 14.7 kDa protein, heat and sodium arsenite, which have been defined previously as inhibitors of cytolysis, inhibit the tumor necrosis factor-alpha (TNF)-induced release of 3H-arachidonic acid from cycloheximide-sensitized C3HA fibroblasts. sodium arsenite 80-95 tumor necrosis factor Homo sapiens 172-199 1417842-1 1992 In this report we show that the adenovirus E3 region 14.7 kDa protein, heat and sodium arsenite, which have been defined previously as inhibitors of cytolysis, inhibit the tumor necrosis factor-alpha (TNF)-induced release of 3H-arachidonic acid from cycloheximide-sensitized C3HA fibroblasts. sodium arsenite 80-95 tumor necrosis factor Homo sapiens 201-204 29479036-11 2018 In conclusion, NaAsO2 exposure might amplify inflammation-related tissue injury by potentiating the apoptosis-inducing effect of TNF-alpha through ROS-dependent mechanism. sodium arsenite 15-21 tumor necrosis factor Homo sapiens 129-138 24961358-5 2015 On the other hand, sodium arsenite activated pro-inflammatory signals, including beta-catenin, nuclear factor-kappaB (NF-kappaB), p38 mitogen-activated protein kinase (MAPK), tumor necrosis factor alpha and cyclooxygenase-2 (COX-2). sodium arsenite 19-34 tumor necrosis factor Homo sapiens 175-202 29479036-3 2018 The effects of excess NaAsO2 on TNF-alpha response and its intracellular signaling are not well understood. sodium arsenite 22-28 tumor necrosis factor Homo sapiens 32-41 29479036-4 2018 We hypothesized that NaAsO2 exposure might affect cellular response to TNF-alpha. sodium arsenite 21-27 tumor necrosis factor Homo sapiens 71-80 26497187-3 2016 Sodium arsenite (1.5 mg/kg, i.p., 2 weeks) abrogated the acetylcholine-induced, endothelium-dependent vasorelaxation by depicting the decrease in serum nitrite/nitrate concentration, reduced glutathione level, and simultaneously enhance the thiobarbituric acid reactive substances (TBARS) level, superoxide level, and tumor necrosis factor-alpha. sodium arsenite 0-15 tumor necrosis factor Homo sapiens 318-345 21281968-4 2011 We report that in vitro pre-treatment of PBMC with sodium arsenite (NaAs) reduces IL2 secretion and T cell proliferation induced by PHA, but does not prevent expression of monocyte-derived cytokines (IL1, IL6, TNFalpha) functioning as lymphocyte-activating factors. sodium arsenite 51-66 tumor necrosis factor Homo sapiens 210-218