PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
11792460-7	2002	The extracellular-regulated protein kinases (ERK) and synaptophysin, proteins implicated in exocytosis, are both tyrosine-phosphorylated by bpV[phen] in a dose- and time-dependent manner, with a maximal effect at 30 microM.	1,10-phenanthroline	144-148	Eph receptor B1	Rattus norvegicus	4-43	
15685932-5	2004	We found that ERK activation could be completely abolished if RINm5F cells were incubated with both bpV (phen) and PD 98059, a specific inhibitor of upstream ERK kinase MEK1.	1,10-phenanthroline	105-109	Eph receptor B1	Rattus norvegicus	14-17	
15685932-5	2004	We found that ERK activation could be completely abolished if RINm5F cells were incubated with both bpV (phen) and PD 98059, a specific inhibitor of upstream ERK kinase MEK1.	1,10-phenanthroline	105-109	Eph receptor B1	Rattus norvegicus	158-161	
11792460-7	2002	The extracellular-regulated protein kinases (ERK) and synaptophysin, proteins implicated in exocytosis, are both tyrosine-phosphorylated by bpV[phen] in a dose- and time-dependent manner, with a maximal effect at 30 microM.	1,10-phenanthroline	144-148	Eph receptor B1	Rattus norvegicus	45-48	
24885237-5	2014	After 3-4 days of IH, extracellular signal-regulated kinases 1/2 (ERK1/2) protein phosphorylation decreased, which could be reversed by superoxide dismutase (SOD), 1,10-phenanthroline (Phe), the PP2A phosphorylation inhibitors, okadaic acid (OKA) and cantharidin, and the ERK phosphorylation activator nicotine (p < 0.05).	1,10-phenanthroline	164-183	Eph receptor B1	Rattus norvegicus	66-69	
8758915-4	1996	N-acetylcysteine and o-phenanthroline abolished ERK activation by BHTOOH, consistent with a requirement for metal-dependent formation of reactive intermediates.	1,10-phenanthroline	21-37	Eph receptor B1	Rattus norvegicus	48-51	
24885237-5	2014	After 3-4 days of IH, extracellular signal-regulated kinases 1/2 (ERK1/2) protein phosphorylation decreased, which could be reversed by superoxide dismutase (SOD), 1,10-phenanthroline (Phe), the PP2A phosphorylation inhibitors, okadaic acid (OKA) and cantharidin, and the ERK phosphorylation activator nicotine (p < 0.05).	1,10-phenanthroline	185-188	Eph receptor B1	Rattus norvegicus	66-69	
24885237-6	2014	In particular, the significantly reduced cell proliferation and increased proportions of cells in the G0/G1 phase after 1-4 days of IH (p < 0.05), which resulted in decreased numbers of PC12 cells, could be reversed by treatment with SOD, Phe, PP2A inhibitors and an ERK activator.	1,10-phenanthroline	242-245	Eph receptor B1	Rattus norvegicus	270-273