PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27941431-6	2017	In addition, circulating levels of miR-134, -10a, -195, -133b were significantly lower and miR-144, -142-5p, -155 were higher (P < 0.05) with development of BOS.	N-[4-(Aminosulfonyl)phenyl]-2-Mercaptobenzamide	160-163	microRNA 144	Homo sapiens	91-98	
25979625-8	2015	RESULTS: Analysis of miR-144 in the biopsy specimens demonstrated 4.1 +- 0.8-fold increases in BOS(+) compared with BOS(-) patients, with a significant reduction in TGIF1 (3.6 +- 1.2-fold), a corepressor of Smads.	N-[4-(Aminosulfonyl)phenyl]-2-Mercaptobenzamide	95-98	microRNA 144	Homo sapiens	21-28	
25979625-8	2015	RESULTS: Analysis of miR-144 in the biopsy specimens demonstrated 4.1 +- 0.8-fold increases in BOS(+) compared with BOS(-) patients, with a significant reduction in TGIF1 (3.6 +- 1.2-fold), a corepressor of Smads.	N-[4-(Aminosulfonyl)phenyl]-2-Mercaptobenzamide	116-119	microRNA 144	Homo sapiens	21-28	
25979625-11	2015	CONCLUSIONS: miR-144 is a critical regulator of the TGF-beta signaling cascade and is over-expressed in lungs with BOS.	N-[4-(Aminosulfonyl)phenyl]-2-Mercaptobenzamide	115-118	microRNA 144	Homo sapiens	13-20