PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26162855-0	2015	Long-term use of indomethacin leads to poor prognoses through promoting the expression of PD-1 and PD-L2 via TRIF/NF-kappaB pathway and JAK/STAT3 pathway to inhibit TNF-alpha and IFN-gamma in hepatocellular carcinoma.	Indomethacin	17-29	programmed cell death 1	Mus musculus	90-94	
26162855-9	2015	In addition, long-term use of indomethacin activates TRIF/NF-kappaB and JAK/STAT3 pathways, and indomethacin promotes the expression of PD-1 and PD-L2 via TRIF/NF-kappaB pathway and JAK/STAT3 pathway respectively in gammadelta T cells.	Indomethacin	96-108	programmed cell death 1	Mus musculus	136-140	
26162855-10	2015	Given these findings, we drew a conclusion that long-term use of indomethacin leads to poor prognoses through promoting the expression of PD-1 and PD-L2 via TRIF/NF-kappaB pathway and JAK/STAT3 pathway to inhibit TNF-alpha and IFN-gamma in HCC.	Indomethacin	65-77	programmed cell death 1	Mus musculus	138-142	
26162855-7	2015	Furthermore, long-term use of indomethacin increased the expression of PD-1 and PD-L2 in programmed death-1 pathway.	Indomethacin	30-42	programmed cell death 1	Mus musculus	71-75	
26162855-8	2015	Blockade of PD-1 and PD-L2 reversed the reduced production of TNF-alpha and IFN-gamma induced by indomethacin in gammadelta T cells.	Indomethacin	97-109	programmed cell death 1	Mus musculus	12-16