PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19926870-9	2010	In COPD lungs, the MC(T) and MC(TC) populations showed alterations in morphology and expression of CD88 (C5a-R), transforming growth factor (TGF)-beta, and renin.	Methylcholanthrene	19-21	transforming growth factor beta 1	Homo sapiens	113-150	
20571097-8	2011	This could be confirmed ex vivo, as the prevalence of non-epitheloid phenotype of MC in the standard group was significantly higher with increasing PD duration and MC isolated from this group showed significantly higher levels of EMT-associated molecules including fibronectin, collagen I, VEGF, IL-8 and TGF-beta levels when compared with the low-GDP group.	Methylcholanthrene	164-166	transforming growth factor beta 1	Homo sapiens	305-313	
19926870-9	2010	In COPD lungs, the MC(T) and MC(TC) populations showed alterations in morphology and expression of CD88 (C5a-R), transforming growth factor (TGF)-beta, and renin.	Methylcholanthrene	29-31	transforming growth factor beta 1	Homo sapiens	113-150	
9054616-5	1997	The number of foci induced by methylcholanthrene, N-methyl-N"-nitro-N-nitrosoguanidine or quercetin was significantly reduced when the cultures were treated with TGF-beta.	Methylcholanthrene	30-48	transforming growth factor beta 1	Homo sapiens	162-170	
15816502-4	2005	TGFbeta is a potent pro-fibrotic factor and induces epithelial-mesenchymal transition (EMT) of the MC.	Methylcholanthrene	99-101	transforming growth factor beta 1	Homo sapiens	0-7	
7882362-11	1995	Some analogues of vitamin D3 (EB 1089, MC 903, and KH 1060) that are known to be potent inhibitors of breast cancer cell growth both in vitro and in vivo had similar or more pronounced inducing effects on TGF-beta 1 mRNA levels.	Methylcholanthrene	39-41	transforming growth factor beta 1	Homo sapiens	205-215	
7808430-5	1994	TGF-beta 1 proved to be the most effective cytokine, because 72 hr of treatment with 2 ng/ml TGF-beta 1 produced nearly 100% inhibition of 3-MC- and benzo(a)pyrene-induced CYP1A1 and CYP1A2 mRNAs and EROD activity.	Methylcholanthrene	139-143	transforming growth factor beta 1	Homo sapiens	0-10	
7808430-5	1994	TGF-beta 1 proved to be the most effective cytokine, because 72 hr of treatment with 2 ng/ml TGF-beta 1 produced nearly 100% inhibition of 3-MC- and benzo(a)pyrene-induced CYP1A1 and CYP1A2 mRNAs and EROD activity.	Methylcholanthrene	139-143	transforming growth factor beta 1	Homo sapiens	93-103	
28179517-5	2017	We demonstrate that a simple change in TGF-beta signalling is sufficient to inhibit MC breakdown during opossum development, indicating that changes in TGF-beta signalling might be key during mammalian evolution.	Methylcholanthrene	84-86	transforming growth factor beta 1	Homo sapiens	39-47	
33475929-2	2021	We investigated if the application of MC at 10 muA/90 s could modulate the expression of remodeling genes transforming growth factor beta (Tgfb), connective tissue growth factor (Ctgf), insulin-like growth factor 1 (Igf1), tenascin C (Tnc), Fibronectin (Fn1), Scleraxis (Scx), Fibromodulin (Fmod) and tenomodulin in NIH/3T3 fibroblasts in a wound healing assay.	Methylcholanthrene	38-40	transforming growth factor beta 1	Homo sapiens	139-143	
34997082-10	2022	MiR-29b inhibits TGF-beta1 induced MMT and replacement of miR-29b in the peritoneal cavity might be effective to prevent development of PM partly through the effects on MC.	Methylcholanthrene	169-171	transforming growth factor beta 1	Homo sapiens	17-26	
28179517-5	2017	We demonstrate that a simple change in TGF-beta signalling is sufficient to inhibit MC breakdown during opossum development, indicating that changes in TGF-beta signalling might be key during mammalian evolution.	Methylcholanthrene	84-86	transforming growth factor beta 1	Homo sapiens	152-160