PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19298066-1	2009	The one-electron-reduced form of vitamin B(12), cob(II)alamin (Co(2+)Cbl), is found in several essential human enzymes, including the cobalamin-dependent methionine synthase (MetH).	Niacinamide	33-42	Cbl proto-oncogene	Homo sapiens	69-72	
15652495-1	2005	Cellular uptake of vitamin B(12) (cobalamin, Cbl) is mediated by a receptor expressed on the plasma membrane that binds transcobalamin (TC) saturated with Cbl and internalizes the TC-Cbl by endocytosis.	Niacinamide	19-28	Cbl proto-oncogene	Homo sapiens	45-48	
15652495-1	2005	Cellular uptake of vitamin B(12) (cobalamin, Cbl) is mediated by a receptor expressed on the plasma membrane that binds transcobalamin (TC) saturated with Cbl and internalizes the TC-Cbl by endocytosis.	Niacinamide	19-28	Cbl proto-oncogene	Homo sapiens	155-158	
15652495-1	2005	Cellular uptake of vitamin B(12) (cobalamin, Cbl) is mediated by a receptor expressed on the plasma membrane that binds transcobalamin (TC) saturated with Cbl and internalizes the TC-Cbl by endocytosis.	Niacinamide	19-28	Cbl proto-oncogene	Homo sapiens	155-158	
15308131-1	2004	Methylmalonic acidemia (MMA) is caused by the deficient activity of l-methylmalonyl-CoA mutase, which is a vitamin B(12) (or cobalamin, Cbl)-dependent enzyme.	Niacinamide	107-116	Cbl proto-oncogene	Homo sapiens	136-139	
21922568-3	2011	We report studies on the kinetics of the reaction between peroxynitrite/peroxynitrous acid and a major intracellular vitamin B(12) form, cob(II)alamin (Cbl(II)), using stopped-flow spectroscopy.	Niacinamide	117-126	Cbl proto-oncogene	Homo sapiens	152-155	
21652039-4	2011	In the present study cob(I)alamin {Cbl(I)}, a reduced form of vitamin B(12), was used for trapping of GA.	Niacinamide	62-71	Cbl proto-oncogene	Homo sapiens	35-38	
21130828-2	2011	The reduced form of vitamin B(12), cob(I)alamin {Cbl(I)}, obtained by reduction of hydroxocobalamin (OH-Cbl) with e.g. sodium borohydride, is one of the most powerful nucleophiles known.	Niacinamide	20-29	Cbl proto-oncogene	Homo sapiens	49-52	
21130828-2	2011	The reduced form of vitamin B(12), cob(I)alamin {Cbl(I)}, obtained by reduction of hydroxocobalamin (OH-Cbl) with e.g. sodium borohydride, is one of the most powerful nucleophiles known.	Niacinamide	20-29	Cbl proto-oncogene	Homo sapiens	104-107	
20876572-1	2010	Vitamin B(12) (cobalamin, Cbl) is essential to the function of two human enzymes, methionine synthase (MS) and methylmalonyl-CoA mutase (MUT).	Niacinamide	0-9	Cbl proto-oncogene	Homo sapiens	26-29	
20089427-1	2010	BACKGROUND: Maternal vitamin B(12) (Cbl) deficiency causes nutritional Cbl deficiency in breastfed infants.	Niacinamide	21-30	Cbl proto-oncogene	Homo sapiens	36-39	
10887099-1	2000	Megaloblastic anemia 1 (MGA1) is an autosomal recessive disorder caused by the selective intestinal malabsorption of intrinsic factor (IF) and vitamin B(12)/cobalamin (Cbl) in complex.	Niacinamide	143-152	Cbl proto-oncogene	Homo sapiens	168-171	
11670948-1	1999	Difluoromethylcobalamin (CF(2)Cbl), a vitamin B(12) analogue with CHF(2) replacing CN, can be synthesized in a two-step procedure from aquocobalamin and CHClF(2).	Niacinamide	38-47	Cbl proto-oncogene	Homo sapiens	30-33	
11670948-6	1999	The packing motif of CF(2)Cbl is very similar to that described for wet vitamin B(12), a distorted hexagonal close packing of the cobalamin, with channels of water running parallel to the crystallographic c axis through the crystal at x = (1)/(4), y = 0, and x = (3)/(4), y = (1)/(2), respectively.	Niacinamide	72-81	Cbl proto-oncogene	Homo sapiens	26-29	
11670948-8	1999	A superposition of the crystal structures of vitamin B(12) and CF(2)Cbl shows a significant change at the molecular level.	Niacinamide	45-54	Cbl proto-oncogene	Homo sapiens	68-71	
20352340-1	2010	Transcobalamin (transcobalamin II, TC) transports plasma vitamin B(12) (cobalamin, Cbl) into cells.	Niacinamide	57-66	Cbl proto-oncogene	Homo sapiens	83-86	
19394404-1	2009	Glial cells, myelin and the interstitium are the structures of the mammalian central nervous system (CNS) mainly affected by vitamin B(12) (cobalamin, Cbl) deficiency.	Niacinamide	125-134	Cbl proto-oncogene	Homo sapiens	151-154	
18606554-1	2008	Derivatives of vitamin B(12) (cobalamin, Cbl) are required for activity of the mitochondrial enzyme L-methylmalonyl-CoA mutase and the cytoplasmic enzyme methionine synthase in human cells.	Niacinamide	15-24	Cbl proto-oncogene	Homo sapiens	41-44