PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
7651731-4	1995	Keratinocytes transfected with the mutant d1787N (which binds to p60, p105, p107 and p300) showed a lethality in response to CDDP (10 micrograms ml-1) fourfold higher than controls and threefold higher in response to DOX and radiation (5 grays).	Cisplatin	125-129	E1A binding protein p300	Mus musculus	85-89	
33327548-0	2020	Inhibition of p300 by Garcinol Protects against Cisplatin-Induced Acute Kidney Injury through Suppression of Oxidative Stress, Inflammation, and Tubular Cell Death in Mice.	Cisplatin	48-57	E1A binding protein p300	Mus musculus	14-18	
33327548-3	2020	However, the role of p300 in cisplatin-induced AKI remains poorly understood.	Cisplatin	29-38	E1A binding protein p300	Mus musculus	21-25	
33327548-10	2020	Taken together, we demonstrated that the inhibition of p300 by garcinol ameliorated cisplatin-induced renal injury, presumably through epigenetic mechanisms.	Cisplatin	84-93	E1A binding protein p300	Mus musculus	55-59	
7651731-8	1995	From these results, we conclude that cell sensitivity to cisplatin and ionizing radiation induced by the E1a oncogene requires binding to p105, p107 and p300 cellular proteins, while sensitivity to Doxorubicin requires binding only to p300.	Cisplatin	57-66	E1A binding protein p300	Mus musculus	153-157	
7651731-8	1995	From these results, we conclude that cell sensitivity to cisplatin and ionizing radiation induced by the E1a oncogene requires binding to p105, p107 and p300 cellular proteins, while sensitivity to Doxorubicin requires binding only to p300.	Cisplatin	57-66	E1A binding protein p300	Mus musculus	235-239