PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9722539-6	1998	UTP, ionomycin, and phorbol ester (phorbol 12-myristate 13-acetate) increased MAP kinase activity and also promoted the tyrosine phosphorylation of RAFTK, the epidermal growth factor (EGF) receptor, SHC, and p120(cbl).	Phorbol Esters	20-33	epidermal growth factor receptor	Rattus norvegicus	159-197	
15284022-5	2004	RIE-1 cells express TACE, and treatment with phorbol ester, an established TACE stimulus, triggered the extracellular release of an EGFR ligand, transforming growth factor-alpha.	Phorbol Esters	45-58	epidermal growth factor receptor	Rattus norvegicus	132-136	
1729394-6	1992	This latter activity was relatively resistant to staurosporine inhibition and phorbol ester treatment, but it phosphorylated the exogenous PKC substrates, histone 1 and the epidermal growth factor receptor peptide KTRLRR.	Phorbol Esters	78-91	epidermal growth factor receptor	Rattus norvegicus	173-205	
17307332-1	2007	While a great deal of attention has been focused on G-protein-coupled receptor (GPCR)-induced epidermal growth factor receptor (EGFR) transactivation, it has been known for many years that the tyrosine kinase activity of the EGFR is inhibited in cells treated with tumor-promoting phorbol esters, a process termed EGFR transmodulation.	Phorbol Esters	281-295	epidermal growth factor receptor	Rattus norvegicus	94-126	
17307332-1	2007	While a great deal of attention has been focused on G-protein-coupled receptor (GPCR)-induced epidermal growth factor receptor (EGFR) transactivation, it has been known for many years that the tyrosine kinase activity of the EGFR is inhibited in cells treated with tumor-promoting phorbol esters, a process termed EGFR transmodulation.	Phorbol Esters	281-295	epidermal growth factor receptor	Rattus norvegicus	128-132	
17307332-1	2007	While a great deal of attention has been focused on G-protein-coupled receptor (GPCR)-induced epidermal growth factor receptor (EGFR) transactivation, it has been known for many years that the tyrosine kinase activity of the EGFR is inhibited in cells treated with tumor-promoting phorbol esters, a process termed EGFR transmodulation.	Phorbol Esters	281-295	epidermal growth factor receptor	Rattus norvegicus	225-229	
17307332-1	2007	While a great deal of attention has been focused on G-protein-coupled receptor (GPCR)-induced epidermal growth factor receptor (EGFR) transactivation, it has been known for many years that the tyrosine kinase activity of the EGFR is inhibited in cells treated with tumor-promoting phorbol esters, a process termed EGFR transmodulation.	Phorbol Esters	281-295	epidermal growth factor receptor	Rattus norvegicus	225-229