PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31714929-7	2019	Overexpression of pink1 or parkin in IFMs can abolish mitochondrial ubiquitination, restore ATP level and extend lifespan, while blocking autophagy via ATG1 knock-down suppress these effects in aged IFMs.	Adenosine Triphosphate	92-95	PTEN-induced putative kinase 1	Drosophila melanogaster	18-23	
32470327-5	2020	Some of these drugs improved the locomotion defects and reduced ATP production caused by PINK1 inactivation in Drosophila and were effective for idiopathic PD-derived neurons with impaired mitochondrial clearance.	Adenosine Triphosphate	64-67	PTEN-induced putative kinase 1	Drosophila melanogaster	89-94	
25611391-8	2015	More importantly, PINK1 ameliorated ATP levels, neuronal integrity and adult fly survival, demonstrating that PINK1 counteracts the neurotoxicity of mHtt.	Adenosine Triphosphate	36-39	PTEN-induced putative kinase 1	Drosophila melanogaster	18-23	
28778978-4	2017	Mitochondrial defects of Pink1 mutant, such as excessive fusion, reduced ATP content, and crista disorganization, were rescued by CHIP but not its ligase-dead mutants.	Adenosine Triphosphate	73-76	PTEN-induced putative kinase 1	Drosophila melanogaster	25-30	
24473149-5	2014	We report that a signaling active version of Ret (Ret(MEN2B) rescues muscle degeneration, disintegration of mitochondria and ATP content of Pink1 mutants.	Adenosine Triphosphate	125-128	PTEN-induced putative kinase 1	Drosophila melanogaster	140-145	
21768365-5	2011	As a consequence, Pink1-KO flies also display a reduced mitochondrial ATP synthesis.	Adenosine Triphosphate	70-73	PTEN-induced putative kinase 1	Drosophila melanogaster	18-23	
20049710-6	2009	The fundamental importance of PINK1 for energy maintenance under increased demand is further corroborated as this deficit can be rescued by adding ATP to the synapse.	Adenosine Triphosphate	147-150	PTEN-induced putative kinase 1	Drosophila melanogaster	30-35