PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24965069-6	2015	S1P and hypotonic extracellular solution induced secretion of ATP from the macrophages, which in both cases was blocked in a similar way by typical VRAC blockers.	Adenosine Triphosphate	62-65	sphingosine-1-phosphate receptor 1	Mus musculus	0-3	
30229580-5	2018	RESULTS: Lack of S1P induced by sphingosine kinase inhibitor (SphKi) treatment caused beta-cell dysfunction and apoptosis, with repression of mitochondrial function shown by decreases in cellular adenosine triphosphate content, the oxygen consumption rate, the expression of oxidative phosphorylation complexes, the mitochondrial membrane potential, and the expression of key regulators of mitochondrial dynamics (mitochondrial dynamin-like GTPase [OPA1] and mitofusin 1 [MFN1]).	Adenosine Triphosphate	196-218	sphingosine-1-phosphate receptor 1	Mus musculus	17-20	
29304533-6	2018	Also, ATP-dependent S1P transport in platelet membrane vesicles containing endogenous MRP4 was inhibited by the MRP inhibitor MK571 and the MRP4-selective compound Ceefourin-1.	Adenosine Triphosphate	6-9	sphingosine-1-phosphate receptor 1	Mus musculus	20-23	
29304533-9	2018	Fluvastatin and rosuvastatin interfered with MRP4 function inhibiting ATP-dependent cGMP (cyclic guanosine monophosphate) uptake into MRP4-containing vesicles, inhibited MRP4-mediated S1P transport in vitro and significantly attenuated endogenous S1P release from agonist-activated platelet ex vivo.	Adenosine Triphosphate	70-73	sphingosine-1-phosphate receptor 1	Mus musculus	184-187	
29304533-9	2018	Fluvastatin and rosuvastatin interfered with MRP4 function inhibiting ATP-dependent cGMP (cyclic guanosine monophosphate) uptake into MRP4-containing vesicles, inhibited MRP4-mediated S1P transport in vitro and significantly attenuated endogenous S1P release from agonist-activated platelet ex vivo.	Adenosine Triphosphate	70-73	sphingosine-1-phosphate receptor 1	Mus musculus	247-250	
29304533-1	2018	Sphingosine-1-phosphate (S1P) is a potent lipid mediator released from activated platelets by an adenosine triphosphate (ATP)-dependent export mechanism.	Adenosine Triphosphate	97-119	sphingosine-1-phosphate receptor 1	Mus musculus	25-28	
29304533-1	2018	Sphingosine-1-phosphate (S1P) is a potent lipid mediator released from activated platelets by an adenosine triphosphate (ATP)-dependent export mechanism.	Adenosine Triphosphate	121-124	sphingosine-1-phosphate receptor 1	Mus musculus	25-28	
29304533-5	2018	Transport studies in membrane vesicles of Sf9 cells containing recombinant human MRP4 revealed that MRP4 mediates ATP-dependent transport of fluorescein- and tritium-labelled S1P.	Adenosine Triphosphate	114-117	sphingosine-1-phosphate receptor 1	Mus musculus	175-178	
24965069-7	2015	We suppose that the S1P-induced ATP secretion in macrophages via activation of VRAC constitutes a functional link between sphingolipid and purinergic signaling in essential processes such as inflammation and migration of leukocytes as well as phagocytosis and the killing of intracellular bacteria.	Adenosine Triphosphate	32-35	sphingosine-1-phosphate receptor 1	Mus musculus	20-23	
15109308-7	2004	Notably, the measurement of S1P formation in vivo by employing labelled ATP revealed that cell-associated SphK activity in the extracellular compartment largely contributed to the transformation of Sph into S1P, with the amount of SphK released into the medium being negligible.	Adenosine Triphosphate	72-75	sphingosine-1-phosphate receptor 1	Mus musculus	28-31	
15459201-1	2004	Sphingosine-1-phosphate (S1P), a lipid signaling molecule that regulates many cellular functions, is synthesized from sphingosine and ATP by the action of sphingosine kinase.	Adenosine Triphosphate	134-137	sphingosine-1-phosphate receptor 1	Mus musculus	25-28	
9371692-4	1997	S1P alone was a weak inducer of DNA synthesis, but its effects were enhanced by phosphocholine (PCho), insulin, ATP or PMA.	Adenosine Triphosphate	112-115	sphingosine-1-phosphate receptor 1	Mus musculus	0-3