PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28725182-5	2017	In peripheral blood mononuclear cells from nAChR gene-deficient mice, we demonstrated that inhibition of ATP-dependent release of IL-1beta by acetylcholine (ACh), nicotine and PC depends on subunits alpha7, alpha9 and alpha10.	Adenosine Triphosphate	105-108	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	43-48	
27349288-1	2016	We demonstrated previously that phosphocholine and phosphocholine-modified macromolecules efficiently inhibit ATP-dependent release of interleukin-1beta from human and murine monocytes by a mechanism involving nicotinic acetylcholine receptors (nAChR).	Adenosine Triphosphate	110-113	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	210-243	
27349288-1	2016	We demonstrated previously that phosphocholine and phosphocholine-modified macromolecules efficiently inhibit ATP-dependent release of interleukin-1beta from human and murine monocytes by a mechanism involving nicotinic acetylcholine receptors (nAChR).	Adenosine Triphosphate	110-113	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	245-250	
23929694-10	2014	Finally, alpha7nAChR activation modulates ATP-induced Ca(2+) response in small intestine resident macrophages.	Adenosine Triphosphate	42-45	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	9-20	
17325648-11	2007	CONCLUSION AND IMPLICATIONS: The nAChR-mediated augmentation of neurogenic contraction is indeed prejunctional, but in the mouse arises from an increase in spontaneous neurotransmitter release that primes smooth muscle for subsequent contraction, while in the guinea pig there is a direct augmentation of evoked neurotransmitter (ATP) release.	Adenosine Triphosphate	330-333	cholinergic receptor, nicotinic, alpha polypeptide 7	Mus musculus	33-38