PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28495429-5	2017	Since, synthesis and activities of selenoenzymes are selenium dependent, we hypothesized that AA-MnSOD cells could have an improvement on antioxidant status undergoing Seleno-L-methionine (SeMet) treatment.	Selenium	53-61	superoxide dismutase 2	Homo sapiens	97-102	
15650415-0	2005	Selenium attenuates expression of MnSOD and uncoupling protein 2 in J774.2 macrophages: molecular mechanism for its cell-death and antiinflammatory activity.	Selenium	0-8	superoxide dismutase 2	Homo sapiens	34-39	
15650415-13	2005	It is therefore concluded that selenium at high nanomolar to low micromolar concentrations shifts the balance between inflammatory response and cell death toward the latter, through a direct effect on the transcription factors Sp1 and NF-kappaB, and down-regulation of MnSOD and UCP2.	Selenium	31-39	superoxide dismutase 2	Homo sapiens	269-274	
11165870-6	2001	Isolated 4.6-fold overexpression of MnSOD activity in stably transfected fibroblasts led to specific resistance from UVA-mediated phototoxicity under selenium-deficient conditions.	Selenium	150-158	superoxide dismutase 2	Homo sapiens	36-41	
25524402-6	2015	The multivariate linear regression adjusting for selenium status showed that plasma Se level was significantly inversely associated only with expression of GSTP1 (beta-coef.=-0.270, p=0.009), PRDXR1 (beta-coef.=-0.245, p=0.017) and SOD2 with an inverse trend toward significance (beta-coef.=-0.186, p=0.074), but without an effect of NRF2 gene variants.	Selenium	84-86	superoxide dismutase 2	Homo sapiens	232-236	
21982398-11	2011	Exploratory analyses of variations in OGG1 and MnSOD genes indicate that hypotheses about patterns of exposure to selenium and smoking combined with data on genetic variation in genes involved in DNA repair can be valuable to pursue.	Selenium	114-122	superoxide dismutase 2	Homo sapiens	47-52	
20477822-2	2011	We previously reported a statistically significant interaction between circulating selenium levels, variants in the superoxide dismutase 2 gene (SOD2; rs4880), and risk of developing prostate cancer and presenting with aggressive prostate cancer.	Selenium	83-91	superoxide dismutase 2	Homo sapiens	145-149	
19074884-3	2008	Selenium status also modifies the effect of the mitochondrial superoxide dismutase (SOD2) SNP Ala16Val on prostate cancer risk.	Selenium	0-8	superoxide dismutase 2	Homo sapiens	84-88	
19528373-2	2009	Our group previously demonstrated a strong interaction between plasma selenium and the manganese superoxide dismutase (SOD2) gene and incident prostate cancer risk.	Selenium	70-78	superoxide dismutase 2	Homo sapiens	87-117	
19528373-2	2009	Our group previously demonstrated a strong interaction between plasma selenium and the manganese superoxide dismutase (SOD2) gene and incident prostate cancer risk.	Selenium	70-78	superoxide dismutase 2	Homo sapiens	119-123	
19528373-3	2009	We now hypothesized that SOD2 modifies the association between selenium level and risk of aggressive prostate cancer at diagnosis.	Selenium	63-71	superoxide dismutase 2	Homo sapiens	25-29	
19528373-7	2009	There was evidence of an interaction between SOD2 and selenium levels such that among men with the AA genotype, higher selenium levels were associated with a reduced risk of presenting with aggressive disease (RR = 0.60; 95% CI, 0.32 to 1.12), whereas among men with a V allele, higher selenium levels were associated with an increased risk of aggressive disease (for VV or VA men, RR = 1.82; 95% CI, 1.27 to 2.61; P for interaction = .007).	Selenium	119-127	superoxide dismutase 2	Homo sapiens	45-49	
19528373-7	2009	There was evidence of an interaction between SOD2 and selenium levels such that among men with the AA genotype, higher selenium levels were associated with a reduced risk of presenting with aggressive disease (RR = 0.60; 95% CI, 0.32 to 1.12), whereas among men with a V allele, higher selenium levels were associated with an increased risk of aggressive disease (for VV or VA men, RR = 1.82; 95% CI, 1.27 to 2.61; P for interaction = .007).	Selenium	119-127	superoxide dismutase 2	Homo sapiens	45-49	
19074884-13	2008	In a low-selenium population, SOD2-Ala16+ men homozygous for SEPP1-Ala234 are at an increased risk of prostate cancer/aggressive prostate cancer especially if ever-smokers, because they are likely to produce more mitochondrial H(2)O(2) that they cannot remove, thereby promoting prostate tumor cell proliferation and migration.	Selenium	9-17	superoxide dismutase 2	Homo sapiens	30-34	
17293063-6	2007	Genes up-regulated by selenium supplementation included TNF, IL1B, IL8, SOD2, CXCL2 and several other immunological and oxidative stress-related genes.	Selenium	22-30	superoxide dismutase 2	Homo sapiens	72-76