PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32279475-0	2020	FFA2 Activation Ameliorates 2,4-Dinitrochlorobenzene-Induced Atopic Dermatitis in Mice.	Dinitrochlorobenzene	28-52	free fatty acid receptor 2	Mus musculus	0-4	
32279475-4	2020	We investigated the efficacy of the FFA2 agonist, 4-chloro-alpha-(1-methylethyl)-N-2-thiazoylylbenzeneacetanilide (4-CMTB), for treatment of atopic dermatitis induced by 2,4-dinitrochlorobenzene (DNCB).	Dinitrochlorobenzene	170-194	free fatty acid receptor 2	Mus musculus	36-40	
32279475-4	2020	We investigated the efficacy of the FFA2 agonist, 4-chloro-alpha-(1-methylethyl)-N-2-thiazoylylbenzeneacetanilide (4-CMTB), for treatment of atopic dermatitis induced by 2,4-dinitrochlorobenzene (DNCB).	Dinitrochlorobenzene	196-200	free fatty acid receptor 2	Mus musculus	36-40	
32279475-10	2020	In conclusion, activation of FFA2 ameliorated DNCB-induced atopic dermatitis, which suggested that FFA2 is a therapeutic target for atopic dermatitis.	Dinitrochlorobenzene	46-50	free fatty acid receptor 2	Mus musculus	29-33	
32279475-10	2020	In conclusion, activation of FFA2 ameliorated DNCB-induced atopic dermatitis, which suggested that FFA2 is a therapeutic target for atopic dermatitis.	Dinitrochlorobenzene	46-50	free fatty acid receptor 2	Mus musculus	99-103