PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12766648-4	2003	Recovery of liver adenosine triphosphate, hepatocellular injury, and expression of glutamine synthetase 1, a gene that is induced by exposure of hepatocytes to low partial pressure of oxygen, were studied at 4 h of resuscitation.	Oxygen	184-190	glutamate-ammonia ligase	Rattus norvegicus	83-105	
12766648-7	2003	Improved hepatocellular oxygen supply was also confirmed by restoration of the physiologic expression pattern of glutamine synthetase 1.	Oxygen	24-30	glutamate-ammonia ligase	Rattus norvegicus	113-135	
2886388-2	1987	In these studies the levels of DNPH-reactive proteins increased steadily during 48 h of continuous oxygen treatment and then decreased to control levels by 54 h. Although the specific activity of two hepatic enzymes, glutamine synthetase and glucose-6-phosphate dehydrogenase, decreased during oxygen treatment, antibody titration of each enzyme indicated that the levels of immunological cross-reactive protein either remained constant or increased slightly during 48 h of oxygen treatment.	Oxygen	99-105	glutamate-ammonia ligase	Rattus norvegicus	217-237	
8866724-1	1996	Exposing 7-day-old rat pups to hypoxia, 8% oxygen/92% nitrogen, for 3 h alters glutamate (GLU), glutamine and glutamine synthetase (GS) activity in the striatum, frontal cortex and hippocampus.	Oxygen	43-49	glutamate-ammonia ligase	Rattus norvegicus	110-130	
1687664-1	1991	We have previously shown that exposure to 100% oxygen for 2 h results in a two-fold decrease in the brain glutamine synthetase activity of neonatal rats.	Oxygen	47-53	glutamate-ammonia ligase	Rattus norvegicus	106-126	
13960585-0	1963	[Effect of oxygen under pressure and methionine sulfoximine on the activity of glutamine synthetase in the rat brain].	Oxygen	11-17	glutamate-ammonia ligase	Rattus norvegicus	79-99	
2886388-2	1987	In these studies the levels of DNPH-reactive proteins increased steadily during 48 h of continuous oxygen treatment and then decreased to control levels by 54 h. Although the specific activity of two hepatic enzymes, glutamine synthetase and glucose-6-phosphate dehydrogenase, decreased during oxygen treatment, antibody titration of each enzyme indicated that the levels of immunological cross-reactive protein either remained constant or increased slightly during 48 h of oxygen treatment.	Oxygen	294-300	glutamate-ammonia ligase	Rattus norvegicus	217-237	
2886388-2	1987	In these studies the levels of DNPH-reactive proteins increased steadily during 48 h of continuous oxygen treatment and then decreased to control levels by 54 h. Although the specific activity of two hepatic enzymes, glutamine synthetase and glucose-6-phosphate dehydrogenase, decreased during oxygen treatment, antibody titration of each enzyme indicated that the levels of immunological cross-reactive protein either remained constant or increased slightly during 48 h of oxygen treatment.	Oxygen	294-300	glutamate-ammonia ligase	Rattus norvegicus	217-237	
31131557-12	2019	The overexpression of GS, GCK, CYP1A, and HIFalpha proteins were observed downstream in the oxygen-poor area.	Oxygen	92-98	glutamate-ammonia ligase	Rattus norvegicus	22-24