PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24193262-1	2013	BACKGROUND AND AIMS: Transketolase-like (TKTL) 1 is one of the key enzymes for anaerobic sugar degradation even in the presence of oxygen (aerobic glycolysis).	Oxygen	131-137	transketolase like 1	Homo sapiens	21-48	
30044385-7	2018	In summary, our results indicate a role of TKTL1 in the adaptation of tumor cells to oxygen deprivation and in the acquisition of radioresistance.	Oxygen	85-91	transketolase like 1	Homo sapiens	43-48	
24304513-3	2013	The transketolase-like protein 1 (TKTL1) represents the enzymatic basis for an anaerobic glucose metabolism even in the presence of oxygen (aerobic glycolysis/Warburg effect), which is concomitant with a more malignant phenotype due to invasive growth/metastasis and resistance to radical and apoptosis inducing therapies.	Oxygen	132-138	transketolase like 1	Homo sapiens	4-32	
24304513-3	2013	The transketolase-like protein 1 (TKTL1) represents the enzymatic basis for an anaerobic glucose metabolism even in the presence of oxygen (aerobic glycolysis/Warburg effect), which is concomitant with a more malignant phenotype due to invasive growth/metastasis and resistance to radical and apoptosis inducing therapies.	Oxygen	132-138	transketolase like 1	Homo sapiens	34-39	
24193262-6	2013	Regulation of TKTL1 under oxygen depletion was analyzed by cultivating cells either in a three-dimensional spheroid model or in a hypoxia incubator chamber.	Oxygen	26-32	transketolase like 1	Homo sapiens	14-19	
21980427-1	2011	BACKGROUND: Transketolase-like 1 (TKTL1) induces glucose degradation through anaerobic pathways, even in presence of oxygen, favoring the malignant aerobic glycolytic phenotype characteristic of tumor cells.	Oxygen	117-123	transketolase like 1	Homo sapiens	12-32	
21980427-1	2011	BACKGROUND: Transketolase-like 1 (TKTL1) induces glucose degradation through anaerobic pathways, even in presence of oxygen, favoring the malignant aerobic glycolytic phenotype characteristic of tumor cells.	Oxygen	117-123	transketolase like 1	Homo sapiens	34-39	
16969476-5	2006	We propose that TKTL1 upregulation is a common phenomenon in GC and cancer of the GEJ leading to an enhanced, oxygen-independent glucose usage which might contribute to a more aggressive tumor growth.	Oxygen	110-116	transketolase like 1	Homo sapiens	16-21	
16465194-7	2006	We propose that TKTL1 upregulation in tumours leads to enhanced, oxygen-independent glucose usage and a lactate-based matrix degradation.	Oxygen	65-71	transketolase like 1	Homo sapiens	16-21