PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29082365-13	2017	CONCLUSION: Continuous exposure of 2 MAC sevoflurane in 2 lit/min O2 simultaneous during prepubertal may create more testicular tissue damage in terms of cellular and molecular function compared to continuous exposure to lower level of sevoflurane by increase in ratio of Bax/Bcl2 and apoptosis in germ cells after puberty.	Oxygen	66-68	BCL2-associated X protein	Mus musculus	272-275	
15604726-0	2004	Bax-induced cell death of Arabidopsis is meditated through reactive oxygen-dependent and -independent processes.	Oxygen	68-74	BCL2-associated X protein	Mus musculus	0-3	
11140697-4	2000	Adult mice exposed to greater than 95% oxygen concentrations for 48 to 88 hours had increased whole-lung mRNA levels of Bax and Bcl-X(L), no change in Bak, Bad, or Bcl-2, and decreased levels of Bcl-w and Bfl-1.	Oxygen	39-45	BCL2-associated X protein	Mus musculus	120-123	
14627695-0	2004	c-Myc sensitization to oxygen deprivation-induced cell death is dependent on Bax/Bak, but is independent of p53 and hypoxia-inducible factor-1.	Oxygen	23-29	BCL2-associated X protein	Mus musculus	77-80	
14627695-10	2004	Thus, oxygen deprivation-induced cell death in fibroblasts with deregulated expression of c-Myc is independent of p53 or HIF-1 status, but is dependent on the Bcl-2 family member Bax or Bak to initiate mitochondrial dependent cell death.	Oxygen	6-12	BCL2-associated X protein	Mus musculus	179-182	
23506274-5	2013	Under 20% oxygen, but not 1.5% or 5%, NGF decreased apoptosis in mouse ovaries by down-regulating the pro-apoptotic genes Bax and p53.	Oxygen	10-16	BCL2-associated X protein	Mus musculus	122-125	
25632145-2	2015	We report here that deficiency in bax exerted broad neuroprotection against excitotoxic injury and oxygen/glucose deprivation in mouse neocortical neuron cultures and reduced infarct size, necrotic injury, and cerebral edema formation after middle cerebral artery occlusion in mice.	Oxygen	99-105	BCL2-associated X protein	Mus musculus	34-37	
22302823-9	2012	Oxygen/glucose deprivation-induced injury in organotypic hippocampal slice cultures confirmed that calpains were specifically activated during bax-dependent apoptosis and in this setting function as downstream cell-death executioners.	Oxygen	0-6	BCL2-associated X protein	Mus musculus	143-146	
22367737-4	2012	Herein, we demonstrate that exposure to atmospheric oxygen rapidly induced p53, TOP2A, and BCL2-associated X protein (BAX) expression and mitochondrial reactive oxygen species (ROS) generation in primary mouse MSCs resulting in oxidative stress, reduced cell viability, and inhibition of cell proliferation.	Oxygen	52-58	BCL2-associated X protein	Mus musculus	91-116	
22367737-4	2012	Herein, we demonstrate that exposure to atmospheric oxygen rapidly induced p53, TOP2A, and BCL2-associated X protein (BAX) expression and mitochondrial reactive oxygen species (ROS) generation in primary mouse MSCs resulting in oxidative stress, reduced cell viability, and inhibition of cell proliferation.	Oxygen	52-58	BCL2-associated X protein	Mus musculus	118-121