PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 8834597-1 1996 Changes in the activity of GOT, GPT, AP and GGTP in blood of rats intoxicated by low doses of N-Nitrosodimethylamine (NDMA) are presented. Dimethylnitrosamine 94-116 glutamic--pyruvic transaminase Rattus norvegicus 32-35 6719461-2 1984 This study demonstrates that these same treatments also potentiate the hepatotoxicity of NDMA as indicated by plasma glutamic pyruvate transaminase (GPT) levels and histologic data. Dimethylnitrosamine 89-93 glutamic--pyruvic transaminase Rattus norvegicus 117-147 6719461-2 1984 This study demonstrates that these same treatments also potentiate the hepatotoxicity of NDMA as indicated by plasma glutamic pyruvate transaminase (GPT) levels and histologic data. Dimethylnitrosamine 89-93 glutamic--pyruvic transaminase Rattus norvegicus 149-152 6719461-3 1984 Pretreatment with acetone or isopropanol (2.5 ml/kg) and 2 days of fasting caused a 2-fold potentiation of NDMA-induced plasma GPT elevation, whereas streptozotocin-induced diabetes caused a 4.6-fold potentiation. Dimethylnitrosamine 107-111 glutamic--pyruvic transaminase Rattus norvegicus 127-130 7191620-2 1980 administration of dimethylnitrosamine (DMN) on 5 consecutive days to rats fed a nutritionally adequate liquid diet resulted 24 hours after the last injection of significant increases in glutamic dehydrogenase (GDH), glutamic oxylacetate transaminase (GOT), and glutamic pyruvate transaminase (GPT) activities in the serum, indicating a striking hepatotoxic effect of this compound. Dimethylnitrosamine 18-37 glutamic--pyruvic transaminase Rattus norvegicus 261-291 7191620-2 1980 administration of dimethylnitrosamine (DMN) on 5 consecutive days to rats fed a nutritionally adequate liquid diet resulted 24 hours after the last injection of significant increases in glutamic dehydrogenase (GDH), glutamic oxylacetate transaminase (GOT), and glutamic pyruvate transaminase (GPT) activities in the serum, indicating a striking hepatotoxic effect of this compound. Dimethylnitrosamine 18-37 glutamic--pyruvic transaminase Rattus norvegicus 293-296 7191620-2 1980 administration of dimethylnitrosamine (DMN) on 5 consecutive days to rats fed a nutritionally adequate liquid diet resulted 24 hours after the last injection of significant increases in glutamic dehydrogenase (GDH), glutamic oxylacetate transaminase (GOT), and glutamic pyruvate transaminase (GPT) activities in the serum, indicating a striking hepatotoxic effect of this compound. Dimethylnitrosamine 39-42 glutamic--pyruvic transaminase Rattus norvegicus 261-291 7191620-2 1980 administration of dimethylnitrosamine (DMN) on 5 consecutive days to rats fed a nutritionally adequate liquid diet resulted 24 hours after the last injection of significant increases in glutamic dehydrogenase (GDH), glutamic oxylacetate transaminase (GOT), and glutamic pyruvate transaminase (GPT) activities in the serum, indicating a striking hepatotoxic effect of this compound. Dimethylnitrosamine 39-42 glutamic--pyruvic transaminase Rattus norvegicus 293-296 24250714-6 2013 Compared with the normal control rats, rats treated with DMN for either 2 weeks or 4 weeks had significantly lower body weights, liver indexes and elevation of hydroxyproline, GOT, and GPT contents. Dimethylnitrosamine 57-60 glutamic--pyruvic transaminase Rattus norvegicus 185-188 15744066-2 2005 After treated with DMN for 4 weeks, the levels of aminotrasferases (GOT, GPT) were significantly elevated in serum, and the levels of total protein (TP) and albumin were significantly decreased in serum and liver homogenates. Dimethylnitrosamine 19-22 glutamic--pyruvic transaminase Rattus norvegicus 73-76