PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25691377-12	2015	A healthy neuron may tolerate free metal toxicity, such as iron in the case of injury-induced amyloid, for as long as twenty years due to this very BACE1 activity.	Iron	59-63	beta-secretase 1	Homo sapiens	148-153	
25934480-6	2015	In cases with brain amyloid pathology (n = 15, age = 78.7 +- 12.7 year), increased BACE1 IR was identified locally at capillaries, arterioles and along the pia, localizing to endothelia, perivascular dystrophic neurites and meningeal cells, and often coexisting with vascular iron deposition.	Iron	276-280	beta-secretase 1	Homo sapiens	83-88	
30776900-0	2019	Iron Dyshomeostasis Induces Binding of APP to BACE1 for Amyloid Pathology, and Impairs APP/Fpn1 Complex in Microglia: Implication in Pathogenesis of Cerebral Microbleeds.	Iron	0-4	beta-secretase 1	Homo sapiens	46-51	
30776900-4	2019	Further results revealed that extracellular iron accumulation might potentially induce binding of APP to BACE1 for amyloid formation and decrease the capability of APP/Fpn1 in mediating iron export.	Iron	44-48	beta-secretase 1	Homo sapiens	105-110