PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 19741202-1 2009 The extent to which the acute phase response (APR) influences iron status indicators in chronic infections is not well documented. Iron 62-66 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 46-49 25714024-0 2015 Dihydroartemisinin and its derivative induce apoptosis in acute myeloid leukemia through Noxa-mediated pathway requiring iron and endoperoxide moiety. Iron 121-125 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 89-93 29343315-1 2018 OBJECTIVE: To assess the impact of the acute-phase response (APR) during inflammation on Fe, Zn and vitamin A biomarkers to allow accurate evaluation of micronutrient status in populations. Iron 89-91 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 61-64 25714024-6 2015 Interfering with the integrity of the endoperoxide moiety of DHA and X-11, as well as chelating intracellular iron with deferoxamine, diminish apoptosis and Noxa induction. Iron 110-114 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 157-161 25714024-8 2015 DHA and X-11 represent a new group of AML cells-apoptosis inducing compounds which work through Noxa up-regulation utilizing the specific endoperoxide moiety and intracellular iron. Iron 176-180 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 96-100 25398733-8 2014 Traditional indicators of iron, zinc, and vitamin A status are altered during the APR, leading to inaccurate estimations of deficiency in populations with a high or unknown prevalence of infection. Iron 26-30 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 82-85 23770082-5 2013 When lysosomal membranes were stabilized by the iron-chelating agent desferrioxamine, H2O2-induced increase in DNA damage, Noxa expression, and subsequent apoptosis were abolished by the inhibition of LMP. Iron 48-52 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 123-127 18506200-1 2009 OBJECTIVES: The acute phase response (APR) influences indicators of iron status. Iron 68-72 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 38-41 19175946-12 2009 It may be possible to develop algorithms, based on the markers of the APR and Fe status, to assess the Fe status among the patients with tuberculosis or other infections eliciting an APR. Iron 78-80 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 183-186 19175946-12 2009 It may be possible to develop algorithms, based on the markers of the APR and Fe status, to assess the Fe status among the patients with tuberculosis or other infections eliciting an APR. Iron 103-105 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 70-73 19175946-12 2009 It may be possible to develop algorithms, based on the markers of the APR and Fe status, to assess the Fe status among the patients with tuberculosis or other infections eliciting an APR. Iron 103-105 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 183-186 18506200-4 2009 We examined whether the APR (measured by AGP) influences the expected relationships between iron status indicators in an HIV-infected population. Iron 92-96 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 24-27 18506200-10 2009 CONCLUSIONS: AGP captured the influence of the APR on iron indicators and their relationships with each other. Iron 54-58 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 47-50 18506200-15 2009 We support the WHO/CDC recommendation that AGP is a useful indicator to assess the influence of the APR on iron status indicators. Iron 107-111 phorbol-12-myristate-13-acetate-induced protein 1 Homo sapiens 100-103