PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 10712402-15 2000 Our observations of increased HDL cholesteryl ester uptake and reduced cellular cholesterol efflux (acute-phase versus native HDL) suggest that displacement of apolipoprotein A-I by SAA results in considerable altered metabolic properties of its main physiological carrier. Cholesterol Esters 34-51 apolipoprotein A-I Oryctolagus cuniculus 160-178