PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25644787-6	2015	In addition, mRNA and protein levels of Akt were increased significantly in the PCB118-treated groups, whereas FoxO3a expression did not show particular variation.	2,3',4,4',5-pentachlorobiphenyl	80-86	AKT serine/threonine kinase 1	Homo sapiens	40-43	
25644787-7	2015	Furthermore, exposure to PCB118 was associated with a significant increase of the protein levels of p-Akt and p-FoxO3a, and these effects were blocked by LY294002.	2,3',4,4',5-pentachlorobiphenyl	25-31	AKT serine/threonine kinase 1	Homo sapiens	102-105	
25644787-10	2015	Our research suggests that PCB118 may induce thyrocyte dysfunction through the Akt/FoxO3a/NIS signalling pathway, which provides potential new insights for finding interventions to counteract the damage to the human body caused by PCBs.	2,3',4,4',5-pentachlorobiphenyl	27-33	AKT serine/threonine kinase 1	Homo sapiens	79-82