PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28795147-3	2017	Here, we evaluated the effects of minimized calcineurin inhibitor (CNI) on DSA development to assess the validity of minimized/withdrawn immunosuppression.	dsa	75-78	calcineurin binding protein 1	Homo sapiens	67-70	
28795147-9	2017	Univariate logistic regression analysis revealed that recipient age at LT younger than 3 years (P = 0.0099) and low CNI (P < 0.0001) were significantly associated with DSA development.	dsa	171-174	calcineurin binding protein 1	Homo sapiens	116-119	
28795147-13	2017	CONCLUSIONS: CNI minimization was an independent risk factor for posttransplant DSA during long-term follow-up after pediatric LT.	dsa	80-83	calcineurin binding protein 1	Homo sapiens	13-16	
28795147-14	2017	Adjusting CNI to appropriate levels is a safe first step to prevent the immunological effects of DSA.	dsa	97-100	calcineurin binding protein 1	Homo sapiens	10-13	
23721554-4	2013	In multivariable modeling, the use of cyclosporine (as opposed to tacrolimus) and low calcineurin inhibitor levels increased the risk of de novo DSA formation, while a calculated MELD score >15 at transplant and recipient age >60 years old reduced the risk.	dsa	145-148	calcineurin binding protein 1	Homo sapiens	86-107