PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33462377-0	2021	DL-3-n-butylphthalide ameliorates diabetes-associated cognitive decline by enhancing PI3K/Akt signaling and suppressing oxidative stress.	3-n-butylphthalide	0-21	thymoma viral proto-oncogene 1	Mus musculus	90-93	
33462377-8	2021	Moreover, we revealed that DL-NBP administration suppressed oxidative stress by upregulating Nrf2/HO-1 signaling, and increased brain-derived neurotrophic factor (BDNF) expression by activating PI3K/Akt/CREB signaling in the hippocampus.	3-n-butylphthalide	27-33	thymoma viral proto-oncogene 1	Mus musculus	199-202	
32066705-0	2020	Dl-3-n-butylphthalide attenuates mouse behavioral deficits to chronic social defeat stress by regulating energy metabolism via AKT/CREB signaling pathway.	3-n-butylphthalide	0-21	thymoma viral proto-oncogene 1	Mus musculus	127-130	
32066705-10	2020	Our findings provide the first evidence showing that NBP can attenuate stress-induced behavioral deficits by modulating energy metabolism by regulating activation of the AKT/CREB signaling pathway.	3-n-butylphthalide	53-56	thymoma viral proto-oncogene 1	Mus musculus	170-173