PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15246640-2	2004	Since ethanolamine is a product of PLD, we hypothesised that an administration of an analogue of ethanolamine, N-methylethanolamine (MEA), decreases PLD activity through a negative feedback mechanism, suppresses collagen accumulation, and thus prevents organ dysfunction.	N-methylaminoethanol	111-131	glycosylphosphatidylinositol specific phospholipase D1	Homo sapiens	35-38	
15246640-0	2004	N-methylethanolamine attenuates cardiac fibrosis and improves diastolic function: inhibition of phospholipase D as a possible mechanism.	N-methylaminoethanol	0-20	glycosylphosphatidylinositol specific phospholipase D1	Homo sapiens	96-111	
15246640-2	2004	Since ethanolamine is a product of PLD, we hypothesised that an administration of an analogue of ethanolamine, N-methylethanolamine (MEA), decreases PLD activity through a negative feedback mechanism, suppresses collagen accumulation, and thus prevents organ dysfunction.	N-methylaminoethanol	111-131	glycosylphosphatidylinositol specific phospholipase D1	Homo sapiens	149-152	
15246640-2	2004	Since ethanolamine is a product of PLD, we hypothesised that an administration of an analogue of ethanolamine, N-methylethanolamine (MEA), decreases PLD activity through a negative feedback mechanism, suppresses collagen accumulation, and thus prevents organ dysfunction.	N-methylaminoethanol	133-136	glycosylphosphatidylinositol specific phospholipase D1	Homo sapiens	35-38	
15246640-2	2004	Since ethanolamine is a product of PLD, we hypothesised that an administration of an analogue of ethanolamine, N-methylethanolamine (MEA), decreases PLD activity through a negative feedback mechanism, suppresses collagen accumulation, and thus prevents organ dysfunction.	N-methylaminoethanol	133-136	glycosylphosphatidylinositol specific phospholipase D1	Homo sapiens	149-152