PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32618019-6	2020	Further research uncovered that Seph induced apoptosis by disrupting mitochondrial homeostasis, inducing mitochondrial depolarization, mitochondrial membrane potential (MMP) loss, and cytochrome c (Cyt c) release and activating c-Jun N-terminal kinase (JNK).	seneciphylline	32-36	mitogen-activated protein kinase 8	Mus musculus	228-251	
32618019-6	2020	Further research uncovered that Seph induced apoptosis by disrupting mitochondrial homeostasis, inducing mitochondrial depolarization, mitochondrial membrane potential (MMP) loss, and cytochrome c (Cyt c) release and activating c-Jun N-terminal kinase (JNK).	seneciphylline	32-36	mitogen-activated protein kinase 8	Mus musculus	253-256	
32618019-7	2020	The Seph-induced apoptosis in hepatocytes could be alleviated by Mdivi-1 (50 muM, a dynamin-related protein 1 inhibitor), as well as SP600125 (25 muM, a specific JNK inhibitor) and ZVAD-fmk (50 muM, a general caspase inhibitor).	seneciphylline	4-8	mitogen-activated protein kinase 8	Mus musculus	162-165