PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34150525-0	2021	Adipose-derived mesenchymal stem cells rescue rat hippocampal cells from aluminum oxide nanoparticle-induced apoptosis via regulation of P53, Abeta, SOX2, OCT4, and CYP2E1.	Aluminum Oxide	73-87	Wistar clone pR53P1 p53 pseudogene	Rattus norvegicus	137-140	
34150525-4	2021	Oral administration of Al2O3-NPs increased the protein levels of P53, cleaved caspase-3, CYP2E1, and beta-amyloid (Abeta); contrarily, AD-MSCs transplantation downregulated the levels of these proteins.	Aluminum Oxide	23-28	Wistar clone pR53P1 p53 pseudogene	Rattus norvegicus	65-68	
32105957-0	2020	Neurotoxicity of aluminum oxide nanoparticles and their mechanistic role in dopaminergic neuron injury involving p53-related pathways.	Aluminum Oxide	17-31	Wistar clone pR53P1 p53 pseudogene	Rattus norvegicus	113-116	
32105957-7	2020	In addition, Nano-Al2O3 decreased the expression of cyclin D1, bcl-2, Mdm2, and phospho-Rb and increased the expression of p53, p21, Bax, and Rb.	Aluminum Oxide	18-23	Wistar clone pR53P1 p53 pseudogene	Rattus norvegicus	123-126	
32105957-8	2020	Therefore, oxidative stress, mitochondrial dysfunction, and p53-related pathways might be important in the process of dopaminergic neurotoxicity induced by Nano-Al2O3.	Aluminum Oxide	161-166	Wistar clone pR53P1 p53 pseudogene	Rattus norvegicus	60-63