PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 20709795-0 2010 Resveratrol prevents light-induced retinal degeneration via suppressing activator protein-1 activation. Resveratrol 0-11 jun proto-oncogene Mus musculus 72-91 24073240-6 2013 The acetylation of c-Jun decreased and its translocation was impeded in the resveratrol-treated T cells. Resveratrol 76-87 jun proto-oncogene Mus musculus 19-24 20709795-8 2010 Retinal activator protein-1 activation, up-regulated following light exposure, was significantly reduced by application of resveratrol. Resveratrol 123-134 jun proto-oncogene Mus musculus 8-27 18551458-0 2008 Pterostilbene is equally potent as resveratrol in inhibiting 12-O-tetradecanoylphorbol-13-acetate activated NFkappaB, AP-1, COX-2, and iNOS in mouse epidermis. Resveratrol 35-46 jun proto-oncogene Mus musculus 118-122 20712904-10 2010 Resveratrol inhibited LPS-induced NF-kappaB activation in both cell types, but inhibited AP-1 activation only in microglia. Resveratrol 0-11 jun proto-oncogene Mus musculus 89-93 18551458-4 2008 Resveratrol and pterostilbene significantly reduced activator protein 1 (AP-1) and NF-kappaB activation. Resveratrol 0-11 jun proto-oncogene Mus musculus 52-71 18551458-4 2008 Resveratrol and pterostilbene significantly reduced activator protein 1 (AP-1) and NF-kappaB activation. Resveratrol 0-11 jun proto-oncogene Mus musculus 73-77 15630167-0 2004 Resveratrol inhibits phorbol ester-induced cyclooxygenase-2 expression in mouse skin: MAPKs and AP-1 as potential molecular targets. Resveratrol 0-11 jun proto-oncogene Mus musculus 96-100 16999939-0 2006 Resveratrol modulates phorbol ester-induced pro-inflammatory signal transduction pathways in mouse skin in vivo: NF-kappaB and AP-1 as prime targets. Resveratrol 0-11 jun proto-oncogene Mus musculus 127-131 15630167-7 2004 In addition, resveratrol prevented TPA-induced DNA binding of activator protein-1 (AP-1). Resveratrol 13-24 jun proto-oncogene Mus musculus 62-81 15630167-7 2004 In addition, resveratrol prevented TPA-induced DNA binding of activator protein-1 (AP-1). Resveratrol 13-24 jun proto-oncogene Mus musculus 83-87 15630167-8 2004 Taken together, suppression of COX-2 expression by blocking the activation of MAPKs and AP-1 may represent possible molecular mechanisms responsible for previously reported anti-tumor promoting effects of resveratrol on mouse skin carcinogenesis. Resveratrol 205-216 jun proto-oncogene Mus musculus 88-92 26100520-4 2015 The combination of ursolic acid + resveratrol inhibited TPA-induced signaling pathways, including EGFR, STAT3, Src, Akt, Cox-2, Fas, NF-kappaB, p38 MAPK, c-Jun, and JNK1/2 while increasing levels of tumor suppressors, such as p21 and PDCD4, to a greater extent compared with the groups treated with the individual compounds. Resveratrol 34-45 jun proto-oncogene Mus musculus 154-159 26100520-7 2015 Furthermore, NF-kappaB, Egr-1, and AP-1 DNA binding activities after TPA treatment were dramatically decreased by the combination of ursolic acid + resveratrol. Resveratrol 148-159 jun proto-oncogene Mus musculus 35-39 10370867-9 1999 As judged by the reverse transcriptase-polymerase chain reaction, resveratrol selectively inhibited TPA-induced expression of c-fos and transforming growth factor-beta 1 (TGF-beta 1), but did not affect other TPA-induced gene products including COX-1, COX-2, c-myc, c-jun, and tumor necrosis factor-alpha. Resveratrol 66-77 jun proto-oncogene Mus musculus 266-271 31844893-0 2020 Resveratrol attenuates diabetes-associated cell centrosome amplification via inhibiting the PKCalpha-p38 to c-myc/c-jun pathway. Resveratrol 0-11 jun proto-oncogene Mus musculus 114-119 31844893-11 2020 Interestingly, resveratrol, but not metformin, was able to attenuate the treatment-induced increase in the levels of p-PKCalpha, p-p38, c-myc, and c-jun, as well as the centrosome amplification. Resveratrol 15-26 jun proto-oncogene Mus musculus 147-152 28511554-6 2017 In the present study, we show that resveratrol could upregulate the expressions of NF-kappaB, IkappaB kinase, JNK, and c-jun in splenic lymphocytes of immunosuppressive mice. Resveratrol 35-46 jun proto-oncogene Mus musculus 119-124