PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31133647-12	2019	trans-Resveratrol and hesperetin combination (tRES-HESP) corrected increased MG and glucose metabolism by increasing expression of Glo1 and decreasing expression of HK-2.	Resveratrol	0-17	hexokinase 2	Homo sapiens	165-169	
27829129-3	2016	Resveratrol impaired hexokinase II (HK2)-mediated glycolysis, and markedly inhibited anchorage-dependent and -independent growth of NSCLC cells.	Resveratrol	0-11	hexokinase 2	Homo sapiens	36-39	
27829129-5	2016	Moreover, we revealed that resveratrol impaired glucose metabolism by mainly inhibiting expression of HK2 mediated by the Akt signaling pathway, and exogenous overexpression of constitutively activated Akt1 in NSCLC cells substantially rescued resveratrol-induced glycolysis suppression.	Resveratrol	27-38	hexokinase 2	Homo sapiens	102-105	
35216280-9	2022	Importantly, glyoxalase 1 inducer-trans-resveratrol and hesperetin in combination (tRES-HESP)-corrected HK2-linked glycolytic overload and unscheduled glycolysis and reversed insulin resistance and improved vascular inflammation in overweight and obese subjects in clinical trial.	Resveratrol	34-51	hexokinase 2	Homo sapiens	104-107	
30445019-9	2019	Dephosphorylated VDAC1 using pretreated resveratrol promoted dissociation with Bax and binding to HK2, which subsequently protected cardiomyocytes against A/R injury.	Resveratrol	40-51	hexokinase 2	Homo sapiens	98-101