PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 22718188-4 2012 Both the urine KIM-1 and Gpnmb to creatinine ratios correlated with the urinary albumin to creatinine ratio, the duration of diabetes, and the stringency of glycemic control but not with blood pressure or baseline eGFR. Creatinine 91-101 hepatitis A virus cellular receptor 1 Homo sapiens 15-20 23988168-8 2013 In this group, 27.3% (3/11) of the cases with normal serum creatinine level showed weakly positive KIM-1 expression in their renal tissues. Creatinine 59-69 hepatitis A virus cellular receptor 1 Homo sapiens 99-104 23837450-6 2013 RESULTS: Urinary KIM-1/creatinine (Cr) was significantly decreased in patients with IgAN after treatment compared to baseline (P < 0.0001, 1.16 [0.51-1.83] vs 0.26 [0.12-0.65] ng/mg). Creatinine 23-33 hepatitis A virus cellular receptor 1 Homo sapiens 17-22 23220287-8 2013 Participants with a combination of low GFR (<60 mL/min/1.72 m(2)) and high KIM-1/creatinine (>128 ng/mmol) had a 3-fold increase in heart failure risk compared with participants with normal GFR and KIM-1 (P < 0.001). Creatinine 84-94 hepatitis A virus cellular receptor 1 Homo sapiens 78-83 23200959-6 2013 RESULTS: Urine geometric mean KIM-1 levels (ng/mg creatinine) were significantly higher in VUR patients than in healthy controls (P=.018). Creatinine 50-60 hepatitis A virus cellular receptor 1 Homo sapiens 30-35 22745276-8 2012 Renal function (analyzed by serum creatinine levels) at the first week posttransplantation correlated with kidney injury molecule-1 and forkhead box P3 mRNA levels. Creatinine 34-44 hepatitis A virus cellular receptor 1 Homo sapiens 107-131 22937100-5 2012 When adjusted for potential confounders, the treatment effect of gentamicin remained significant only for KIM-1 (mean difference from not treated, 1.35 ng/mg urinary creatinine; 95% CI 0.05-2.65). Creatinine 166-176 hepatitis A virus cellular receptor 1 Homo sapiens 106-111 21970770-19 2011 Urinary KIM-1 expression may be detected after exposure to a variety of nephrotoxic agents, even when serum creatinine concentrations do not increase, and this has been accepted by regulatory authorities as a sensitive biomarker of acute kidney injury during early drug development. Creatinine 108-118 hepatitis A virus cellular receptor 1 Homo sapiens 8-13 21467131-12 2011 Univariate analysis showed that baseline serum creatinine and urinary excretion of total protein, alpha(1)m, beta(2)m, immunoglobulin G, KIM-1 and NGAL were significantly associated with ESRD. Creatinine 47-57 hepatitis A virus cellular receptor 1 Homo sapiens 137-142 21846754-5 2011 Urinary analysis showed that KIM-1 was significantly elevated in heart failure patients compared with healthy controls (1100, IQR 620-1920 vs. 550, IQR 320-740 ng/g urinary creatinine, P < 0.001). Creatinine 173-183 hepatitis A virus cellular receptor 1 Homo sapiens 29-34 21402675-7 2011 Urinary levels of KIM-1 in IgAN positively correlated with levels of serum creatinine and proteinuria and negatively with creatinine clearance. Creatinine 75-85 hepatitis A virus cellular receptor 1 Homo sapiens 18-23 21402675-7 2011 Urinary levels of KIM-1 in IgAN positively correlated with levels of serum creatinine and proteinuria and negatively with creatinine clearance. Creatinine 122-132 hepatitis A virus cellular receptor 1 Homo sapiens 18-23 21168598-12 2010 Upon univariate analysis urinary KIM-1 strongly correlated with serum creatinine (r = .64) and eGFR (r = -.71), and only weakly with other parameters. Creatinine 70-80 hepatitis A virus cellular receptor 1 Homo sapiens 33-38 21996181-10 2011 Their expressions positively correlated with serum creatinine concentrations after 6 months after transplantation: LCN2 (r=.65, P<.0001), HAVCR1 (r=.44, P=.006). Creatinine 51-61 hepatitis A virus cellular receptor 1 Homo sapiens 141-147 21996225-5 2011 Upon univariate analysis, urinary KIM-1 strongly correlated with serum creatinine (r=.54) and eGFR (r=.66) but only weakly with other parameters. Creatinine 71-81 hepatitis A virus cellular receptor 1 Homo sapiens 34-39 19522876-8 2009 Multiple regression analysis showed that urinary KIM-1 at brain death diagnosis was a positive predictor of recipient serum creatinine, 14 days (p < 0.001) and 1 year (p < 0.05) after kidney transplantation. Creatinine 124-134 hepatitis A virus cellular receptor 1 Homo sapiens 49-54 18165774-10 2007 High KIM-1 excretion was associated with proteinuria, low creatinine clearance, and high donor age (all P<0.01). Creatinine 58-68 hepatitis A virus cellular receptor 1 Homo sapiens 5-10 18160964-5 2008 In this group, higher KIM-1 staining predicted a better outcome with improved blood urea nitrogen (BUN), serum creatinine, and estimated glomerular filtration rate (eGFR) over an ensuing 18 months. Creatinine 111-121 hepatitis A virus cellular receptor 1 Homo sapiens 22-27 18160964-8 2008 KIM-1 expression significantly correlated with serum creatinine and BUN, and inversely with the eGFR on the biopsy day. Creatinine 53-63 hepatitis A virus cellular receptor 1 Homo sapiens 0-5 18493238-3 2008 KIM-1 levels showed positive correlation with serum creatinine and blood urea nitrogen levels, and inverse correlation with estimated glomerular filtration rate. Creatinine 52-62 hepatitis A virus cellular receptor 1 Homo sapiens 0-5 18165774-11 2007 In multivariate Cox regression analyses, prediction of graft loss by KIM-1 appeared independent of creatinine clearance, proteinuria, and donor age. Creatinine 99-109 hepatitis A virus cellular receptor 1 Homo sapiens 69-74 33771116-14 2021 Similarly, urinary KIM-1 excretion increased in both groups (DeltaKIM-1: IS 115.8 [74.1; 156.2] ng/mmol creatinine, p < 0.001 vs. PL 152.4 [120.1; 307.9] ng/mmol creatinine, p < 0.001). Creatinine 104-114 hepatitis A virus cellular receptor 1 Homo sapiens 19-24 15744000-7 2005 Urinary hKIM-1 was normalized to the urinary creatinine concentration (U(Cr)). Creatinine 45-55 hepatitis A virus cellular receptor 1 Homo sapiens 8-14 34355651-0 2021 Diagnostic performance of kidney injury molecule-1 for detection of abnormal urinary albumin-to-creatinine ratio in type 2 diabetes mellitus. Creatinine 96-106 hepatitis A virus cellular receptor 1 Homo sapiens 26-50 34729216-5 2021 Results: The median concentration of urinary KIM-1 normalized to urinary creatinine was lower in patients with RCC compared to UC (1.35 vs 1.86 ng/mg creatinine, p = 0.04). Creatinine 73-83 hepatitis A virus cellular receptor 1 Homo sapiens 45-50 34729216-5 2021 Results: The median concentration of urinary KIM-1 normalized to urinary creatinine was lower in patients with RCC compared to UC (1.35 vs 1.86 ng/mg creatinine, p = 0.04). Creatinine 150-160 hepatitis A virus cellular receptor 1 Homo sapiens 45-50 35419185-9 2022 Compared with before treatment, after 3 months of intervention, the levels of serum uric acid, urine microalbumin/creatinine, urine NGAL/creatinine, and urine KIM-1/creatinine were significantly decreased in the febuxostat group (P < 0.05), while the changes of blood urea nitrogen, serum creatinine, and epidermal growth factor receptor (eGFR) were not statistically significant (P > 0.05). Creatinine 165-175 hepatitis A virus cellular receptor 1 Homo sapiens 159-164 35635326-6 2022 KIM-1 and MCP-1 were standardized by urinary creatinine. Creatinine 45-55 hepatitis A virus cellular receptor 1 Homo sapiens 0-5 32487784-9 2020 The serum KIM-1 level positively correlated with microalbuminuria and serum creatinine (r = 0.596, P = 0.001 and r = 0.402, P = 0.034, respectively). Creatinine 76-86 hepatitis A virus cellular receptor 1 Homo sapiens 10-15 35419185-11 2022 After 3 months of intervention, compared with the control group, the serum uric acid, microalbumin/creatinine, urine NGAL/creatinine, and urine KIM-1/creatinine were significantly decreased in the febuxostat group (P < 0.05), but there was no significant difference in blood urea nitrogen, creatinine, and eGFR (P > 0.05). Creatinine 150-160 hepatitis A virus cellular receptor 1 Homo sapiens 144-149 32841436-5 2020 Urine-values of NAG and KIM-1 got related to urinary creatinine. Creatinine 53-63 hepatitis A virus cellular receptor 1 Homo sapiens 24-29 33272156-11 2021 Area under the curve for receiver operating characteristic showed that accuracy of serum creatinine (0.267; 95% CI, 0.11-0.43) at day 0 of cyclosporine treatment was significantly lower (P = .017) than the accuracy of urine creatinine (0.477; 95% CI, 0.28-0.67) and urine levels of the KIM-1 kidney injury molecule (0.594; 95% CI, 0.41-0.78). Creatinine 89-99 hepatitis A virus cellular receptor 1 Homo sapiens 286-291 33140691-2 2021 The aim of this study is to describe the behavior of KIM-1 (Kidney Injury Molecule) and NGAL (Neutrophil Gelatinase Associated Lipocalin) as early predictors of renal damage, comparing them with serum creatinine and creatinine clearance, in neonates undergoing cardiac surgery. Creatinine 216-226 hepatitis A virus cellular receptor 1 Homo sapiens 53-58 31483944-8 2020 In a multivariate logistic model, both serum and urinary KIM-1 were associated with an increased albumin-to-creatinine ratio (>30 mg/g Cr), but only the serum KIM-1 was associated with a lower eGFR (<60 mL/min/1.73 m2 ), after adjustment for covariates. Creatinine 108-118 hepatitis A virus cellular receptor 1 Homo sapiens 57-62 31538126-7 2019 Results: There was a moderate negative correlation between the percentage change of creatinine values and the percentage change of KIM-1 values (r = -0.357, p = 0.016). Creatinine 84-94 hepatitis A virus cellular receptor 1 Homo sapiens 131-136 32541154-2 2020 Whether urinary KIM-1 indexed for creatinine (uKIM-1/Cr) is a prognostic marker of disease progression in ADPKD is unknown.In this secondary analysis of a prospective cohort study, we sought to determine whether patients with high as opposed to low uKIM-1/CR at baseline had greater rates of eGFR loss and height-adjusted total kidney volume (HtTKV) increase. Creatinine 34-44 hepatitis A virus cellular receptor 1 Homo sapiens 16-21 31646460-3 2019 KIM-1 staining scores were significantly correlated with serum creatinine (sCr) levels (P < 0.05) in all age groups. Creatinine 63-73 hepatitis A virus cellular receptor 1 Homo sapiens 0-5 30989103-7 2019 Urinary KIM-1 was normalized with urine creatinine concentration. Creatinine 40-50 hepatitis A virus cellular receptor 1 Homo sapiens 8-13 31372130-9 2019 Moderate positive association were seen for KIM1 with creatinine levels (r=0.530; p<0.001), and HbA1c (r=0.576; p<0.001) in all patients. Creatinine 54-64 hepatitis A virus cellular receptor 1 Homo sapiens 44-48 31372130-11 2019 Multiple logistic regression controlling for age showed that KIM1 was independently associated with BUN (r=0.727; p<0.001), creatinine (r=0.510; p<0.001) and HbA1c (r=0.401; p=0.008) in all groups. Creatinine 127-137 hepatitis A virus cellular receptor 1 Homo sapiens 61-65 30989103-11 2019 The creatinine normalized urinary KIM-1 was significantly higher when urine protein was 1 + to 3+ (0.75-5 g/L). Creatinine 4-14 hepatitis A virus cellular receptor 1 Homo sapiens 34-39 31966633-10 2017 There was a significant positive correlation of total score of KIM-1 expression and the parameters of kidney function for AKI, including serum creatinine (Cr) and blood urea nitrogen (BUN). Creatinine 143-153 hepatitis A virus cellular receptor 1 Homo sapiens 63-68 28884615-6 2017 Treatment with kidney injury molecule-1-Res NPs significantly reduced creatinine and protected against tubulointerstitial injury in a murine model of CKD. Creatinine 70-80 hepatitis A virus cellular receptor 1 Homo sapiens 15-39 30110569-3 2018 In adult kidneys ( n = 52 subjects), we found that the intensity of KIM-1 staining significantly correlated with the antemortem level of serum creatinine, and this was independent of the extent of tissue autolysis. Creatinine 143-153 hepatitis A virus cellular receptor 1 Homo sapiens 68-73 30180818-9 2018 Furthermore KIM-1 data showed a significant increase 2 days before serum creatinine rise in comparison to the corresponding KIM-1 levels in patients who developed AKI (P = 0.001). Creatinine 73-83 hepatitis A virus cellular receptor 1 Homo sapiens 12-17 28103129-8 2017 KIM-1 was the most sensitive marker with a cut-off of 170.4 pg/mg creatinine (sensitivity 91.4%, specificity 59.1%), whereas CA19-9 was the most specific with a cut-off of 51.3 U/mg creatinine (sensitivity 48.9%, specificity 88.0%). Creatinine 66-76 hepatitis A virus cellular receptor 1 Homo sapiens 0-5 28979926-5 2017 RESULTS: Urinary KIM-1 and ANG concentrations were strongly correlated to each other and were significantly and independently associated with cold ischemia time, delayed graft function, and plasma creatinine 10 days after transplantation, indicating that these markers reflect the severity of IRI. Creatinine 197-207 hepatitis A virus cellular receptor 1 Homo sapiens 17-22 31966633-10 2017 There was a significant positive correlation of total score of KIM-1 expression and the parameters of kidney function for AKI, including serum creatinine (Cr) and blood urea nitrogen (BUN). Creatinine 155-157 hepatitis A virus cellular receptor 1 Homo sapiens 63-68 28103909-10 2017 IGFBP1, insulin-like growth factor binding protein 3 (IGFBP3), fibronectin 1 (FN1) and KIM1 showed significant correlations with serum creatinine, and IGFBP1, KIM1 and kallikrein 1 (KLK1) with eGFR (p < 0.05). Creatinine 135-145 hepatitis A virus cellular receptor 1 Homo sapiens 87-91 28584738-0 2017 Performance Characteristics of Kidney Injury Molecule-1 In Relation to Creatinine, Urea, and Microalbuminuria in the Diagnosis of Kidney Disease. Creatinine 71-81 hepatitis A virus cellular receptor 1 Homo sapiens 31-55 28584738-3 2017 AIMS: This study was conducted to determine the diagnostic value of serum creatinine, urea, and microalbuminuria (MAU) in relation to the novel biomarker, KIM-1. Creatinine 74-84 hepatitis A virus cellular receptor 1 Homo sapiens 155-160 28584738-8 2017 Logistic regression analysis showed that creatinine and urea (R2 = 0.75 and R2 = 0.72, respectively, P < 0.001 for both) were better predictors of kidney disease than MAU (R2 = 0.64, P < 0.001) and KIM-1 (R2 = 0.046, P = 0.116). Creatinine 41-51 hepatitis A virus cellular receptor 1 Homo sapiens 204-209 27852120-7 2017 RESULTS: Urinary levels of KIM-1 significantly decreased after hydronephrosis treatment in patients with unilateral obstruction (1.19 ng/mL vs 0.76 ng/mL creatinine, p = 0.002), additionally KIM-1 was significantly higher in patients with hydronephrosis compared to stone disease patients without radiological evidence of obstruction (1.19 vs 0.64, p = 0.006). Creatinine 154-164 hepatitis A virus cellular receptor 1 Homo sapiens 27-32 27852120-9 2017 We found that a KIM-1 value of 0.735 ng/mg creatinine had a sensitivity of 75% and specificity of 67% to predict the presence of hydronephrosis in preoperative studies (95% CI 0.58-0.87, p = 0.006). Creatinine 43-53 hepatitis A virus cellular receptor 1 Homo sapiens 16-21 28596840-5 2017 The pronounced changes in serum creatinine were accompanied by increased urinary excretion of beta-2-microglobulin and KIM1. Creatinine 32-42 hepatitis A virus cellular receptor 1 Homo sapiens 119-123 25923545-9 2016 RESULTS: KIM-1 levels were found to be 0.86+-0.48 ng/mg creatinine in the patient, and 0.71+-0.46 pg/mL in the control groups (P>0.05). Creatinine 56-66 hepatitis A virus cellular receptor 1 Homo sapiens 9-14 27132791-1 2016 BACKGROUND: This study was designed to determine the diagnostic role of urinary kidney injury molecule (KIM)-1 levels in renal damage in patients with type 2 diabetes mellitus according to the urinary albumin/creatinine ratio. Creatinine 209-219 hepatitis A virus cellular receptor 1 Homo sapiens 104-110 27132791-8 2016 Positive correlation was found between KIM-1 and urine microalbumin-urine microalbumin/creatinine (r = 0.479 P < 0.001; r = 0. Creatinine 87-97 hepatitis A virus cellular receptor 1 Homo sapiens 39-44 27605629-8 2016 The creatinine increases were accompanied by a rise in the levels of urinary renal damage markers [beta-2-microglobulin (B2M), total protein, kidney injury molecule (KIM1), alpha-glutathione S-transferase (aGST), N-acetyl-beta-(d)-glucosaminidase (NAG)]. Creatinine 4-14 hepatitis A virus cellular receptor 1 Homo sapiens 166-170 27966578-4 2016 We retrospectively analyzed 92 workers exposed to occupational lead and demonstrated a better correlation ship between blood lead levels and urine excretion of KIM-1 than other traditional renal injury biomarkers following creatinine adjustment. Creatinine 223-233 hepatitis A virus cellular receptor 1 Homo sapiens 160-165 27642395-7 2016 KIM-1 showed a weak negative correlation with creatinine (r = -0.279, p = 0.09), whereas MAU was positively correlated with creatinine in participants with kidney disease with statistical significance (r = 0.556, p = 0.001). Creatinine 46-56 hepatitis A virus cellular receptor 1 Homo sapiens 0-5 27270293-7 2016 RESULTS: In pooled, multivariable-adjusted analyses, log-transformed, creatinine-normalized urinary KIM-1 levels were higher in those with lower eGFR {beta = -0.03 per 10 mL/min/1.73 m(2) [95% confidence interval (CI) -0.05 to -0.02]} and greater albuminuria [beta = 0.16 per unit of log albumin:creatinine ratio (95% CI 0.15-0.17)]. Creatinine 70-80 hepatitis A virus cellular receptor 1 Homo sapiens 100-105 27270293-7 2016 RESULTS: In pooled, multivariable-adjusted analyses, log-transformed, creatinine-normalized urinary KIM-1 levels were higher in those with lower eGFR {beta = -0.03 per 10 mL/min/1.73 m(2) [95% confidence interval (CI) -0.05 to -0.02]} and greater albuminuria [beta = 0.16 per unit of log albumin:creatinine ratio (95% CI 0.15-0.17)]. Creatinine 296-306 hepatitis A virus cellular receptor 1 Homo sapiens 100-105 26795762-6 2016 RESULTS: Preoperative urine KIM-1/Cr ratio was higher than postoperative 14th day. Creatinine 34-36 hepatitis A virus cellular receptor 1 Homo sapiens 28-33 26795762-9 2016 CONCLUSION: In renal stone patients, the elevated urine KIM-1/Cr ratio levels increase further according to stone size. Creatinine 62-64 hepatitis A virus cellular receptor 1 Homo sapiens 56-61 26795762-10 2016 KIM-1/Cr ratio is a promising marker might be helpful in monitoring the damage created by stone disease. Creatinine 6-8 hepatitis A virus cellular receptor 1 Homo sapiens 0-5 26538632-8 2016 mTOR pathway inhibition in KIM-1-overexpressing transgenic mice also significantly ameliorated serum creatinine level, proteinuria, tubular injury, and kidney inflammation. Creatinine 101-111 hepatitis A virus cellular receptor 1 Homo sapiens 27-32 26511274-6 2016 In chronic HF, plasma KIM-1 was associated with GFR (P < 0.001), creatinine, and cystatin C. Creatinine 68-78 hepatitis A virus cellular receptor 1 Homo sapiens 22-27 26511274-10 2016 In acute HF, higher plasma KIM-1 levels were associated with higher creatinine, lower albumin, and presence of diabetes. Creatinine 68-78 hepatitis A virus cellular receptor 1 Homo sapiens 27-32 27858892-6 2016 The area under the receiver operating characteristic curve (AUC-ROC) of serum KIM-1 for identifying renal dysfunction in all enrolled patients was 0.94 (95% confidence interval [95% CI], 0.87 to 1.02; P < 0.001), while the AUC-ROC of serum creatinine was only 0.82 (95% CI, 0.60 to 1.03; P < 0.01).Serum KIM-1 is a promising new diagnostic biomarker of renal dysfunction during long-term ADV therapy for CHB patients. Creatinine 243-253 hepatitis A virus cellular receptor 1 Homo sapiens 78-83 27321055-6 2016 RESULTS: In a multivariable Cox regression model adjusting for age, glomerular filtration rate, and urinary albumin/creatinine ratio, higher urinary KIM-1/creatinine was associated with an increased risk for cardiovascular mortality (HR per SD increase 1.51, 95% confidence intervals 1.03-2.24, P = 0.03). Creatinine 116-126 hepatitis A virus cellular receptor 1 Homo sapiens 149-154 27321055-6 2016 RESULTS: In a multivariable Cox regression model adjusting for age, glomerular filtration rate, and urinary albumin/creatinine ratio, higher urinary KIM-1/creatinine was associated with an increased risk for cardiovascular mortality (HR per SD increase 1.51, 95% confidence intervals 1.03-2.24, P = 0.03). Creatinine 155-165 hepatitis A virus cellular receptor 1 Homo sapiens 149-154 27321055-8 2016 CONCLUSION: In elderly men with diabetes, higher urinary KIM-1/creatinine was associated with an increased long-term risk of cardiovascular mortality independently of established markers of diabetic nephropathy. Creatinine 63-73 hepatitis A virus cellular receptor 1 Homo sapiens 57-62 26751954-8 2016 Correction of urine VDBP concentrations for creatinine excretion confirmed its predictive value and was consistent with increased levels of urinary Kidney Injury Molecule-1 (KIM-1) and baseline plasma creatinine in patients with above mentioned complications. Creatinine 44-54 hepatitis A virus cellular receptor 1 Homo sapiens 174-179 26248472-7 2015 Median values of urinary KIM-1 were higher in patients with AKI than in those without AKI [10.7, interquartile range (IQR) 1.6-17.9 vs. 4.3 (IQR 1.3-6.1) ng/mg creatinine; p < 0.01]. Creatinine 160-170 hepatitis A virus cellular receptor 1 Homo sapiens 25-30 27296256-10 2016 Clusterin, MCP-1 and KIM-1 positively and significantly correlated with serum creatinine at the week following the determination of BMs in the multivariate linear regression models adjusted for other confounders. Creatinine 78-88 hepatitis A virus cellular receptor 1 Homo sapiens 21-26 24904085-10 2014 In a cohort of patients with type 1 diabetes and proteinuria, serum KIM-1 level at baseline strongly predicted rate of eGFR loss and risk of ESRD during 5-15 years of follow-up, after adjustment for baseline urinary albumin-to-creatinine ratio, eGFR, and Hb1Ac. Creatinine 227-237 hepatitis A virus cellular receptor 1 Homo sapiens 68-73 26350438-11 2015 CONCLUSIONS: Elevated urinary KIM-1/Cr may be associated with a higher risk of incident ESRD, but it does not add to risk prediction after accounting for traditional markers of kidney function in this population. Creatinine 36-38 hepatitis A virus cellular receptor 1 Homo sapiens 30-35 26407820-6 2015 RESULTS: The urinary levels normalized with urine creatinine of KIM-1 and MCP-1, but not NGAL, NAG, and beta2-microglobulin in AKI (+) samples were significantly higher than those in AKI (-) samples. Creatinine 50-60 hepatitis A virus cellular receptor 1 Homo sapiens 64-69 25784666-4 2015 KIM-1 was measured at baseline and normalized with urinary creatinine. Creatinine 59-69 hepatitis A virus cellular receptor 1 Homo sapiens 0-5 25316431-4 2015 HRs were reported per SD of creatinine (Cr)-normalised log-transformed KIM-1, NAG and NGAL, and for three categories of L-FABP. Creatinine 40-42 hepatitis A virus cellular receptor 1 Homo sapiens 71-76 24656453-5 2014 PREDICTORS: Ratios of urine KIM-1, IL-18, and albumin to creatinine (KIM-1:Cr, IL-18:Cr, and ACR, respectively). Creatinine 57-67 hepatitis A virus cellular receptor 1 Homo sapiens 69-74 24923577-7 2014 Higher urinary KIM-1/creatinine (from 24-hour urine collections) was associated with a higher risk for cardiovascular mortality (hazard ratio per SD increase, 1.27; 95% confidence interval [95% CI], 1.05 to 1.54; P=0.01). Creatinine 21-31 hepatitis A virus cellular receptor 1 Homo sapiens 15-20 24598218-14 2014 The number of t-Kim-1 cells at baseline was significantly correlated with the estimated glomerular filtration rate (R = 0.72; P = 0.005) and serum creatinine (R = 0.53; P = 0.005) after 6-8 months of treatment. Creatinine 147-157 hepatitis A virus cellular receptor 1 Homo sapiens 16-21