PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17951357-5	2008	Because endoglin suppresses PCa cell motility in an activin-like kinase receptor-2 (ALK2)- and Smad1-dependent manner, we sought to determine whether genistein was activating the ALK2-Smad1 pathway.	Genistein	150-159	activin A receptor type 1	Homo sapiens	179-183	
17951357-6	2008	Although treatment with genistein or overexpression of Smad1 or ALK2 all increased Smad1-responsive promoter activity and decreased cell motility, genistein"s efficacy was abrogated by either Smad1 or ALK2 knockdown.	Genistein	147-156	activin A receptor type 1	Homo sapiens	64-68	
17951357-6	2008	Although treatment with genistein or overexpression of Smad1 or ALK2 all increased Smad1-responsive promoter activity and decreased cell motility, genistein"s efficacy was abrogated by either Smad1 or ALK2 knockdown.	Genistein	147-156	activin A receptor type 1	Homo sapiens	201-205