PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15928241-10	2005	CONCLUSIONS: Our data suggest that the phenotype associated with POU1F1 mutations may be more variable, with the occasional preservation of TSH secretion.	Thyrotropin	140-143	POU class 1 homeobox 1	Homo sapiens	65-71	
7892156-0	1994	Clinical and immunohistochemical studies on TSH-secreting pituitary adenoma: its multihormonality and expression of Pit-1.	Thyrotropin	44-47	POU class 1 homeobox 1	Homo sapiens	116-121	
8834538-6	1996	The highest incidence was observed in 15 GH-secreting adenomas and 8 TSH-secreting adenomas, in which Pit-1 mRNA was detected in all cases.	Thyrotropin	69-72	POU class 1 homeobox 1	Homo sapiens	102-107	
7852536-9	1995	Consequently, the current description of Pit-1 gene mutations leading to complete GH, PRL, and TSH deficiencies needs to be expanded to GH and PRL deficiencies associated with a compromise of the thyrotroph"s ability to synthesize TSH.	Thyrotropin	95-98	POU class 1 homeobox 1	Homo sapiens	41-46	
7892156-13	1994	From our investigations, the role of Pit-1 in multidirectional differentiation during the development of TSH-secreting adenoma was suggested.	Thyrotropin	105-108	POU class 1 homeobox 1	Homo sapiens	37-42	
12780757-9	2003	A previously described heterozygous Arg271Trp mutation in exon 6 of the POU1F1 gene was identified in a female infant who presented with growth failure and was diagnosed with TSH then GH deficiencies.	Thyrotropin	175-178	POU class 1 homeobox 1	Homo sapiens	72-78	
11081207-2	1999	In the human pituitary adenomas, Pit-1 is highly expressed in GH secreting and TSH secreting adenomas as it can well be anticipated.	Thyrotropin	79-82	POU class 1 homeobox 1	Homo sapiens	33-38	
32138434-1	1993	Pit-1, the pituitary-specific transcriptional factor, has been known as a gene that regulates the functional differentiation of the anterior pituitary gland, especially in GH, PRL, and TSH production.	Thyrotropin	185-188	POU class 1 homeobox 1	Homo sapiens	0-5	
8077321-12	1993	These data demonstrate selective expression of the human pit-1 gene in adenohypophysial cell types responsible for GH, PRL, and/or TSH synthesis and are consistent with a predominantly pretranslational regulatory mechanism for Pit-1 expression in the human.	Thyrotropin	131-134	POU class 1 homeobox 1	Homo sapiens	57-62	
8350517-7	1993	Sequence comparisons using PCR amplified PIT1 gene sequences revealed only one nonsense mutation in the patient, and established that this alteration caused the combined deficiencies of TSH, GH and PRL.	Thyrotropin	186-189	POU class 1 homeobox 1	Homo sapiens	41-45